When da² or Flacc^A heterozygous mothers are crossed to Sxl^fP7B0/Y fathers only 53% and 38%, respectively, of the expected Sxl^fP7B0/+ female progeny survive to adulthood.

(Guo et al., 2018)

Sxl^fP7B0;msl-2²²⁷/Df(2L)Exel7016 females do not display any decrease in viability compared to controls. Ime4^Δ22-3;Sxl^fP7B0 double heterozygous daughters from Ime4^Δ22-3 mutant females show strongly reduced female viability and tumorous ovaries (no decrease in viability or ovarian defects are observed in the presence of maternal wild-type copy of Ime4). The reduced viability phenotype can be fully rescued by combination with msl-2²²⁷/Df(2L)Exel7016 although these females frequently show signs of sexual transformation and display male-specific features such as sex-combs, which are mosaic to various degrees. Sxl^fP7B0;YT521-B^MI02006 double heterozygous daughters from YT521-B^MI02006 mutant females show reduced viability and sexual transformations such as male abdominal pigmentation.

(Haussmann et al., 2016)

Ime4^null/+, Sxl^fP7B0/+ females, but not males, exhibit severely reduced survival to hatching, as compared to controls.

(Lence et al., 2016)

Removing one copy of Rbp9^P2690 during oogenesis in combination with zygotic heterozygosity for Sxl^fP7B0 results in female lethality. No female lethality is seen in the reverse cross. This female lethality is suppressed when msl-3¹ is also zygotically removed.

(Zaharieva et al., 2015)

Females trans-heterozygous for Sxl^fP7B0 and dm¹ are approximately two-thirds as viable as their dm¹/+ siblings. Females trans-heterozygous for Sxl^fP7B0 and dm^P0 are approximately two-thirds as viable as their dm^P0/+ siblings. Females trans-heterozygous for Sxl^fP7B0 and dm^P1 are approximately two-thirds as viable as their dm^P1/+ siblings. Sxl^{N.Hsp83.T:Ecol\lacZ} females that are trans-heterozygous for Sxl^fP7B0 and dm¹ are two-thirds as viable as their Sxl^{N.Hsp83.T:Ecol\lacZ} Sxl^fP7B0/+ sisters. Transgenic females trans-heterozygous for Df(1)dm75e19 and Sxl^fP7B0 are only half as viable as Sxl^fP7B0 female siblings.

(Kappes et al., 2011)

Sxl^fP7B0 suppresses the semi-lethality of homozygous cav²²⁴⁸ males. In progeny from Su(var)205⁵ heterozygous females crossed to Sxl^fP7B0/Y males, female viability is dramatically reduced. The effect of Su(var)205⁵ is strictly maternal; no significant reduction in female viability is observed in the reciprocal cross. In progeny from Su(var)205⁴ heterozygous females crossed to Sxl^fP7B0/Y males, female viability is dramatically reduced. In progeny from Su(var)205² heterozygous females crossed to Sxl^fP7B0/Y males, female viability is reduced.

(Li et al., 2011)

The viability of Sxl^fP7B0/+ females is significantly reduced if they also carry one maternally-derived copy of msl-1^L60, msl-2¹ or msl-3¹. The viability of Sxl^fP7B0/+ females is not affected if they also carry one maternally-derived copy of mle¹. The almost complete lethality of Sxl^fP7B0/+ females carrying a maternally-derived copy of msl-3¹ is significantly rescued (to 98.7% viability) by a maternally-derived copy of Sxl^M1.

(Gladstein et al., 2010)

The female progeny of fl(2)d¹/+ females mated to Sxl^fP7B0/Y males show only 65.3% viability. The female progeny of fl(2)d²/+ females mated to Sxl^fP7B0/Y males show only 87.4% viability. The female progeny of snf^J210/+ females mated to Sxl^fP7B0/Y males show only 83.6% viability. The female progeny of snf^J210/+ ; fl(2)d¹/+ females mated to Sxl^fP7B0/Y males show only 27.6% viability. The female progeny of snf^J210/+ ; fl(2)d²/+ females mated to Sxl^fP7B0/Y males show only 74.4% viability. The female progeny of snf¹⁴⁸/+ females mated to Sxl^fP7B0/Y males show only 48.0% viability. The female progeny of snf¹⁴⁸/+ ; fl(2)d¹/+ females mated to Sxl^fP7B0/Y males show only 11.1% viability. The female progeny of snf¹⁴⁸/+ ; fl(2)d²/+ females mated to Sxl^fP7B0/Y males show only 45.6% viability. The female progeny of snf^J210/+ ; snf^5MER females mated to Sxl^fP7B0/Y males show 98% viability. The female progeny of snf^J210/+ ; fl(2)d¹/+ ; snf^5MER females mated to Sxl^fP7B0/Y males show 16.1% viability. The female progeny of snf^J210/+ ; fl(2)d²/+ ; snf^5MER females mated to Sxl^fP7B0/Y males show 79.3% viability. The female progeny of U2af38^ΔE18/+ females mated to Sxl^fP7B0/Y males are completely viable. The female progeny of U2af38^ΔE18/fl(2)d¹ females mated to Sxl^fP7B0/Y males show only 25.8% viability. The female progeny of U2af38^ΔE18/fl(2)d² females mated to Sxl^fP7B0/Y males show only 90.1% viability.

(Penn et al., 2008)

If snf¹⁴⁸/snf¹⁴⁸, Sxl^cF1.otu females are crossed with Sxl^fP7B0 males only 6% of her Sxl^fP7B0/+ daughters survive. When mothers heterozygous for both snf^J210 and snRNP70K⁰²¹⁰⁷ are crossed with Sxl^fP7B0 males, a significant reduction in viability is seen in Sxl^fP7B0/+ daughters. When mothers heterozygous for both snf^J210 and U2af38^ΔE18 are crossed with Sxl^fP7B0 males, a significant reduction in viability is seen in Sxl^fP7B0/+ daughters. When mothers heterozygous for both snf^J210 and U2af50^XR15 are crossed with Sxl^fP7B0 males, no significant reduction in viability is seen in Sxl^fP7B0/+ daughters.

(Nagengast et al., 2003)

The preferential sensitivity of males to the lethal effects of expression of run^Scer\UAS.cLa under the control of Scer\GAL4^nos.PG is not suppressed by Sxl^fP7B0.

(Tracey et al., 2000)

When embryos are also mutant for nos^BN, the germ cells form two clusters instead of the one they form in single mutants for nos^BN.

(Deshpande et al., 1999)

Female Sxl^fP7B0/+ embryos are fully viable if their mothers are heterozygous for da², 80% viable if their mothers are heterozygous for snf^J210, but only 7% viable if their mothers are heterozygous for both da² and snf^J210. The weak interaction between Sxl^fP7B0 and snf^J210 is enhanced by Df(2R)Su(z)3-1.iy, Aats-asp^TW6, Aats-asp^TW3, Aats-asp^12-21R, Aats-asp¹ or Aats-asp^25-1R; the viability of female Sxl^fP7B0/+ embryos derived from mothers heterozygous for snf^J210 and one of Df(2R)Su(z)3-1.iy, Aats-asp^TW6, Aats-asp^TW3, Aats-asp^12-21R, Aats-asp¹ or Aats-asp^25-1R is reduced. The weak interaction between Sxl^fP7B0 and snf^J210 is enhanced by Aats-trp⁰³⁵⁵⁹, Aats-trp⁴⁴²⁰, Aats-trp^P-74, Aats-trp^P-28B or Df(3R)by10; the viability of female Sxl^fP7B0/+ embryos derived from mothers heterozygous for snf^J210 and one of Aats-trp⁰³⁵⁵⁹, Aats-trp⁴⁴²⁰, Aats-trp^P-74, Aats-trp^P-28B or Df(3R)by10 is reduced.

(Stitzinger et al., 1999)

Lethal in female double heterozygous combination with fl(1)35³⁵. Females doubly heterozygous for fl(1)35⁴⁶ and Sxl^fP7B0 show 2.1% viability. Lethality of fl(1)35⁴⁶/Sxl^fP7B0 females can occur at embryonic or larval stages. The viability of Sxl^fP7B0 in female double heterozygous combination with l(1)43⁴³ is reduced.

(Anand et al., 1998)

Female lethal-synergistic interactions with snf^- mutations.

(Salz and Flickinger, 1996)

Heterozygous females from snf^e8H mothers die, with same phenotype as shown by dying Sxl mutants. This phenotype can be suppressed if the embryo carries the constitutive Sxl^M4 or Sxl^M1 allele.

(Albrecht and Salz, 1993)

The number of vitellogenic eggs produced by ovo^D2 heterozygous females is increased by Sxl^fP7B0.

(Oliver et al., 1990)