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General Information
Symbol
Dmel\trxE2
Species
D. melanogaster
Name
FlyBase ID
FBal0017174
Feature type
allele
Associated gene
Associated Insertion(s)
Carried in Construct
Mutagen
Nature of the Allele
Mutations Mapped to the Genome
 
Type
Location
Additional Notes
References
Associated Sequence Data
DNA sequence
Protein sequence
 
 
Progenitor genotype
Cytology
Nature of the lesion
Statement
Reference
Expression Data
Reporter Expression
Additional Information
Statement
Reference
 
Marker for
Reflects expression of
Reporter construct used in assay
Human Disease Associations
Disease Ontology (DO) Annotations
Models Based on Experimental Evidence ( 0 )
Disease
Evidence
References
Modifiers Based on Experimental Evidence ( 0 )
Disease
Interaction
References
Comments on Models/Modifiers Based on Experimental Evidence ( 0 )
 
Phenotypic Data
Phenotypic Class
Phenotype Manifest In
Detailed Description
Statement
Reference

trxE2/trxE2 somatic clones show similar levels of intestinal stem cell proliferation as controls.

trxE2 mutant somatic clones show a growth disadvantage compared to the wild-type cells as the trxE2 mutant tissue is relatively underrepresented in the mosaic adult eyes. Eyes composed almost entirely of the mutant tissue (generated by mitotic recombination with a chromosome bearing a recessive cell-lethal mutation) are much smaller than in controls. trxE2 homozygous clones in the third instar larval eye discs display high levels of apoptosis (detected by Casp3 antibody staining).

53.9% of heterozygous females have ectopic wing veins.

Developmental apoptosis is trxE2 embryos is similar to wild type.

trxE2 flies show a haltere to wing transformation.

Homozygous mutant clones in the central region of the eye disc cause a delay in photoreceptor differentiation, but clones at the posterior or lateral margins of the disc show a strong loss of photoreceptor differentiation accompanied by overgrowth.

Larval cuticles of trxE2 somatic clone mutant embryos show only mild homeotic transformations.

trxE2/+ enhances the telomeric position effect (TPE) of P{hsp26-pt-T}39C-27. trxE2/+ has no effect on the telomeric position effect (TPE) of P{hsp26-pt-T}39C-5 or P{wA}4-4.

trx1/trxE2 flies show transformations of wing to haltere; 57% show strong transformation, 34% show weak transformation and 9% show no transformation.

Heterozygotes do not have a held-out wing phenotype.

Transformation of abdominal segment A6 to A5.

No effect on the eye pigment phenotype of wT81.

Pairing sensitive repression is unchanged in iab-7 PRE lines that carry trxE2.

Heterozygous adults sometimes display homeotic transformation of thoracic and abdominal segments.

Mutation changes the level of w expression in ph-plac+3 flies; eye colour is lighter.

Suppressor of the Dlw1/Dlw+ ventral to dorsal wing transformation.

trxE2, brm2 double heterozygotes frequently display homeotic transformations: fifth abdominal segment is transformed to a more anterior identity and haltere to wing.

dominant; suppresses extra sex combs in Pc4/+

External Data
Interactions
Show genetic interaction network for Enhancers & Suppressors
Phenotypic Class
Enhanced by
Statement
Reference

brm2, trx[+]/trxE2 has visible | dominant phenotype, enhanceable by Bap60[+]/Bap601

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by JIL-1[+]/JIL-1z2

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by Asx[+]/Asx13

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by Asx3/Asx[+]

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by E(z)[+]/E(z)5

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by Psc[+]/Psc1

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by Sce[+]/SceD1

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by Su(z)21/Su(z)2[+]

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by Su(z)2Arp1/Su(z)2[+]

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by Su(z)4[+]/Su(z)41

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by Su(z)6[+]/Su(z)61

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by Su(z)7[+]/Su(z)71

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by Trl[+]/TrlR85

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by lidk06801/lid[+]

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by sxc[+]/sxc1

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by E(Pc)[+]/E(Pc)1

brm2, trx[+]/trxE2 has visible | dominant | homeotic phenotype, enhanceable by lawcp1/lawc[+]

brm2, trx[+]/trxE2 has visible | dominant | homeotic phenotype, enhanceable by lawc[+]/lawcEF520

brm2, trx[+]/trxE2 has visible | dominant | homeotic phenotype, enhanceable by Df(1)RA2/+

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by lawcp1/lawc[+]

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by lawc[+]/lawcEF520

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by Df(1)RA2/+

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by mod(mdg4)T16/mod(mdg4)[+]

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by mod(mdg4)[+]/mod(mdg4)ul

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, enhanceable by mod(mdg4)[+]/mod(mdg4)T6

brm2, trx[+]/trxE2 has visible | dominant | homeotic phenotype, enhanceable by mod(mdg4)T16/mod(mdg4)[+]

brm2, trx[+]/trxE2 has visible | dominant | homeotic phenotype, enhanceable by mod(mdg4)[+]/mod(mdg4)ul

brm2, trx[+]/trxE2 has visible | dominant | homeotic phenotype, enhanceable by mod(mdg4)[+]/mod(mdg4)T6

Suppressed by
Statement
Reference

trxE2 has visible | dominant phenotype, suppressible | partially by EloA[+]/EloAG4930

trxE2 has visible | dominant phenotype, suppressible | partially by EloCSH1520/l(2)SH1520[+]

trxE2 has visible | dominant phenotype, suppressible | partially by Elongin-C[+]/EloCSH1299

brm[+]/brm2, trxE2 has homeotic | dominant phenotype, suppressible by CG9007[+]/upSETe00365

brm[+]/brm2, trxE2 has homeotic | dominant phenotype, suppressible by upSETMB08950/CG9007[+]

trxE2/trx1 has visible | homeotic phenotype, suppressible | partially by Pp1-87B[+]/Pp1-87B87Bg-6

trxE2/trx1 has visible | homeotic phenotype, suppressible | partially by Pp1-87B[+]/Pp1-87B1

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, suppressible by dom14/dom[+]

trxE2 has visible | homeotic phenotype, suppressible by dom14/dom[+]

brm2, trx[+]/trxE2 has visible | dominant | homeotic phenotype, suppressible by dom14/dom[+]

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, suppressible by M(2)21AB[+]/Sam-S1

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, suppressible by Pc3/Pc[+]

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, suppressible by Pcl7/Pcl[+]

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, suppressible by esc10/esc[+]

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, suppressible by esc21/esc[+]

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, suppressible by esc9/esc[+]

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, suppressible by mxc[+]/mxc1

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, suppressible by mxc[+]/mxcM1

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, suppressible by ph-d[+]/ph-d503

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, suppressible by phob/pho[+]

brm[+]/brm2, trxE2 has visible | dominant | homeotic phenotype, suppressible by sxc[+]/sxc4

NOT suppressed by
Statement
Reference

trxE2 has visible | homeotic phenotype, non-suppressible by Df(2R)ED3921

Enhancer of
Statement
Reference

trx[+]/trxE2 is an enhancer of visible | dominant phenotype of brm[+]/brm2, taraL4

trx[+]/trxE2 is an enhancer of eye color defective phenotype of wYGfPfMG

trx[+]/trxE2 is an enhancer of visible | homeotic phenotype of Dsp11

trxE2 is an enhancer of visible | homeotic phenotype of brm2, lawcp1

trx[+], trxE2, brm[+], brm2 is an enhancer of visible | homeotic phenotype of lawcp1

NOT Enhancer of
Statement
Reference
Suppressor of
Statement
Reference

trx[+]/trxE2 is a suppressor of visible | dominant phenotype of gcmPyx

trxE2 is a suppressor of visible | homeotic phenotype of esc6

NOT Suppressor of
Statement
Reference
Other
Phenotype Manifest In
Enhanced by
Statement
Reference

brm2, trx[+]/trxE2 has humeral bristle phenotype, enhanceable by Bap60[+]/Bap601

brm[+]/brm2, trxE2 has adult abdominal segment 5 | ectopic | male phenotype, enhanceable by JIL-1[+]/JIL-1z2

brm[+]/brm2, trxE2 has adult abdominal segment 6 | male phenotype, enhanceable by JIL-1[+]/JIL-1z2

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, enhanceable by Asx[+]/Asx13

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, enhanceable by Asx3/Asx[+]

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, enhanceable by E(z)[+]/E(z)5

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, enhanceable by Psc[+]/Psc1

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, enhanceable by Sce[+]/SceD1

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, enhanceable by Su(z)21/Su(z)2[+]

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, enhanceable by Su(z)2Arp1/Su(z)2[+]

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, enhanceable by Su(z)4[+]/Su(z)41

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, enhanceable by Su(z)6[+]/Su(z)61

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, enhanceable by Su(z)7[+]/Su(z)71

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, enhanceable by Trl[+]/TrlR85

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, enhanceable by lidk06801/lid[+]

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, enhanceable by sxc[+]/sxc1

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, enhanceable by E(Pc)[+]/E(Pc)1

brm[+]/brm2, trxE2 has haltere phenotype, enhanceable by lawcp1/lawc[+]

brm[+]/brm2, trxE2 has metathoracic leg phenotype, enhanceable by lawcp1/lawc[+]

trxE2 has phenotype, enhanceable by Snr1R3

NOT Enhanced by
Statement
Reference

trxE2 has phenotype, non-enhanceable by Alhunspecified

Suppressed by
Statement
Reference

trxE2 has wing vein | ectopic phenotype, suppressible | partially by EloA[+]/EloAG4930

trxE2 has wing vein | ectopic phenotype, suppressible | partially by EloCSH1520/l(2)SH1520[+]

trxE2 has wing vein | ectopic phenotype, suppressible | partially by Elongin-C[+]/EloCSH1299

brm[+]/brm2, trxE2 has metathoracic leg phenotype, suppressible by CG9007[+]/upSETe00365

brm[+]/brm2, trxE2 has metathoracic leg phenotype, suppressible by upSETMB08950/CG9007[+]

trxE2/trx1 has wing phenotype, suppressible | partially by Pp1-87B[+]/Pp1-87B87Bg-6

trxE2/trx1 has haltere | ectopic phenotype, suppressible | partially by Pp1-87B[+]/Pp1-87B87Bg-6

trxE2/trx1 has wing phenotype, suppressible | partially by Pp1-87B[+]/Pp1-87B1

trxE2/trx1 has haltere | ectopic phenotype, suppressible | partially by Pp1-87B[+]/Pp1-87B1

brm[+]/brm2, trxE2 has abdominal segment 6 phenotype, suppressible by dom14/dom[+]

brm[+]/brm2, trxE2 has haltere phenotype, suppressible by dom14/dom[+]

brm2, trx[+]/trxE2 has abdominal segment 6 phenotype, suppressible by dom14/dom[+]

brm2, trx[+]/trxE2 has haltere phenotype, suppressible by dom14/dom[+]

trxE2 has abdominal segment 6 phenotype, suppressible by dom14/dom[+]

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, suppressible by M(2)21AB[+]/Sam-S1

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, suppressible by Pc3/Pc[+]

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, suppressible by Pcl7/Pcl[+]

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, suppressible by esc10/esc[+]

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, suppressible by esc21/esc[+]

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, suppressible by esc9/esc[+]

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, suppressible by mxc[+]/mxc1

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, suppressible by mxc[+]/mxcM1

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, suppressible by ph-d[+]/ph-d503

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, suppressible by phob/pho[+]

brm[+]/brm2, trxE2 has adult metathoracic segment phenotype, suppressible by sxc[+]/sxc4

NOT suppressed by
Statement
Reference

trxE2 has haltere phenotype, non-suppressible by Df(2R)ED3921

trxE2 has wing | ectopic phenotype, non-suppressible by Df(2R)ED3921

trxE2 has phenotype, non-suppressible by Alhunspecified

Enhancer of
Statement
Reference

trx[+]/trxE2 is an enhancer of eye phenotype of Hsap\MECP2UAS.FL, Scer\GAL4GMR.PU

trx[+]/trxE2 is an enhancer of eye phenotype of Hsap\MECP2Δ166.UAS, Scer\GAL4GMR.PU

trxE2 is an enhancer of wing phenotype of sdFab-X

trx[+]/trxE2 is an enhancer of wing phenotype of brm[+]/brm2, taraL4

trx[+]/trxE2 is an enhancer of pigment cell phenotype of wYGfPfMG

trx[+]/trxE2 is an enhancer of haltere phenotype of Dsp11

trx[+]/trxE2 is an enhancer of wing | ectopic phenotype of Dsp11

trx[+], trxE2, brm[+], brm2 is an enhancer of arista phenotype of lawcp1

trxE2 is an enhancer of arista phenotype of brm2, lawcp1

trx[+], trxE2, brm[+], brm2 is an enhancer of phenotype of mod(mdg4)ul

NOT Enhancer of
Statement
Reference

trx[+]/trxE2 is a non-enhancer of wing vein | adult stage phenotype of Marcal1UAS.cBa, Scer\GAL4Tub.PU

trxE2 is a non-enhancer of wing vein | ectopic phenotype of Snr1R3/Snr1E1

trxE2 is a non-enhancer of eye phenotype of Scer\GAL4ey.PH, osaUAS.cCa

Suppressor of
Statement
Reference

trx[+]/trxE2 is a suppressor of chaeta | supernumerary phenotype of gcmPyx

trxE2 is a suppressor of eye phenotype of CycEJP

trxE2 is a suppressor of phenotype of Abd-BFab7-P18.1

trxE2 is a suppressor of phenotype of Pc4

NOT Suppressor of
Statement
Reference

trx[+]/trxE2 is a non-suppressor of wing vein | adult stage phenotype of Marcal1UAS.cBa, Scer\GAL4Tub.PU

trxE2 is a non-suppressor of eye phenotype of Scer\GAL4ey.PH, brmK804R.UAS.Tag:HA

trxE2 is a non-suppressor of wing vein | ectopic phenotype of Snr1R3/Snr1E1

trxE2/brm2 is a non-suppressor of wing phenotype of Scer\GAL4OK386, nejUAS.cMa

trxE2 is a non-suppressor of eye phenotype of Scer\GAL4ey.PH, osaUAS.cCa

Other
Statement
Reference

brm2, trx[+]/trxE2 has wing phenotype

brm[+]/brm2, tara1/tara[+], trxE2 has wing phenotype

brm[+]/brm2, taraL4/tara[+], trxE2 has wing phenotype

brm[+]/brm2, tara1, trx[+]/trxE2 has wing phenotype

brm2, tara1/tara[+], trx[+]/trxE2 has wing phenotype

brm2, taraL4/tara[+], trx[+]/trxE2 has wing phenotype

Additional Comments
Genetic Interactions
Statement
Reference

The formation of ectopic wing veins observed in adult flies expressing Marcal1Scer\UAS.cBa under the control of Scer\GAL4tub.PU is not affected by combination with a single copy of trxE2.

The penetrance of the ectopic wing vein phenotype seen in trxE2/+ females (53.9% penetrance) is reduced if they are also heterozygous for one of EloAG4930 (10.4% penetrance), EloCSH1520 (5.4%) or EloCSH1299 (16.7%).

56% of brm2 trxE2 double heterozygotes have an apical bristle on the third leg (usually found on the second leg). Heterozygosity for either upSETe00365 or upSETMB08950 reduces the penetrance of this phenotype to 9% or 4% respectively.

7.7% of Rm6201086/trxE2 double heterozygous males show transformation of abdominal segment A6 into A5. 6.6% of the double heterozygotes show transformation of haltere into wing.

The increased apoptosis seen in Scer\GAL4nub-AC-62 RYBPScer\UAS.cBa wing discs is diminished in a trxE2/+ background.

21% of fs(1)h4/+;trxE2/+ adults show segmental deletions, and 41% show transformations of haltere to wing and T1 or T3 leg and plura to T2 identity.

52% of RYBPKG08683/+ ; trxE2/+ males show depigmentation of the fifth abdominal segment. 95% of RYBPKG08683/RYBPKG08683 ; trxE2/+ males show depigmentation of the fifth abdominal segment.

Df(2R)ED3921 fails to suppress the haltere to wing transformation of trxE2 flies.

trxE2/+ largely rescues the wing defects seen in RYBPScer\UAS.cBa; Scer\GAL4sd-SG29.1 animals.

5% of brm2 trxE2 double heterozygotes show loss of humeral bristles. The penetrance of this phenotype is increased to 15% by Bap601/+.

Bap601/+ ; trxE2/+ double heterozygotes do not show loss of humeral bristles.

trxE2 esc6 double mutants show partial suppression of homeotic transformations in embryonic/first instar larval cuticle (i.e. thoracic denticle belts are comparable to those found in wild-type embryos), but the head is abnormal and abdominal segments are still partly transformed towards the eighth abdominal segment.

Pp1-87B87Bg-6 dominantly suppresses the transformation of wing to haltere seen in trx1/trxE2 flies; 36% show strong transformation, 42% show weak transformation and 22% show no transformation.

Pp1-87B1 dominantly suppresses the transformation of wing to haltere seen in trx1/trxE2 flies; 31% show strong transformation, 39% show weak transformation and 30% show no transformation.

Expression of Pp1-87BScer\UAS.T:Ivir\HA1 under the control of Scer\GAL4arm.PS has no effect on the transformation of wing to haltere seen in trx1/trxE2 flies.

brm2 trxE2 double heterozygous males show a low frequency of A6 to A5 transformation; the frequency is 9% if brm2 trxE2 is maternally derived and 17% if brm2 trxE2 is paternally derived. The frequency of A6 to A5 transformation seen in brm2 trxE2 double heterozygous males is increased if they also carry JIL-1z2; the frequency is 65% if JIL-1z2 is maternally derived and brm2 trxE2 is paternally derived and is 75% in the reciprocal cross.

Mutant weakly suppresses the CycEJP rough eye phenotype.

2% of trxE2 taraL4 double heterozygotes have a held-out wing phenotype. 1% of trxE2 tara1 double heterozygotes have a held-out wing phenotype. 5% of trxE2 brm2 double heterozygotes have a held-out wing phenotype. 45% of trxE2 brm2 taraL4 triple heterozygotes have a held-out wing phenotype. 38% of trxE2 brm2 tara1 triple heterozygotes have a held-out wing phenotype.

The frequency of transformation of halteres into wings seen in Dsp11/Y flies is enhanced by trxE2/+. The enhancement is dramatically reduced if the female parents are heterozygous rather than homozygous for Dsp11.

The abdominal segment A6 to A5 transformation and the haltere to wing transformation of brm2/+, trxE2/+ is dominantly suppressed by dom14.

35.3% of trxE2 brm2 double heterozygotes show T3 to T2 transformations. The penetrance of this phenotype is enhanced by one copy of lidk06801, sxc1, SceD1, E(z)5, TrlR85, Su(z)41, Su(z)71, Su(z)61, E(Pc)1, Su(z)21, Su(z)2Arp1, Psc1, Asx3 or Asx13 and suppressed by one copy of Pc3, ph-d503, Pcl7, phob, mxcM1, mxc1, esc9, esc10, esc21, sxc4 or M(2)21AB1.

Mutant heterozygotes show a moderate enhancement of the eye phenotype see in P{UAS-lacZ.Abd-B.5F24} leading to lighter eyes. In trxE2/+, Scer\GAL4hs.PB, wFab-7.5F24 flies, an embryonic heat shock pulse leads to eyes lighter than with wFab-7.5F24 alone.

The addition of trxE2 does not affect the variable eye size phenotype seen in osaScer\UAS.cCa/Scer\GAL4ey.PH flies.

Has little or no effect on the phenotype of E2fGMR.PD DpGMR.PD BacA\p35GMR.PH flies.

brm2 trxE2 double heterozygotes show partial transformation of the 5th abdominal segment to fourth abdominal segment, but do not hold out their wings at any significantly higher frequency than that seen in brm2/+ single heterozygotes.

The frequency and severity of homeotic transformations (such as haltere to wing or third leg to second leg transformations) in brm2 trxE2 double heterozygotes is dominantly enhanced by lawcp1. The enhancement is stronger in a lawcp1 maternal background. The arista to leg transformation characteristic of lawcp1 is also enhanced in these flies. The frequency of homeotic transformations in brm2 trxE2 double heterozygotes is dominantly enhanced by lawcEF520 or Df(1)RA2.

10.0% of trxE2 brm2 double heterozygous adults show a homeotic phenotype. This is increased to 56.5% if the flies are also heterozygous for mod(mdg4)T16, 24.9% if the flies are also heterozygous for mod(mdg4)ul and 35.7% if the flies are also heterozygous for mod(mdg4)T6. The combination brm2 trxE2 dominantly enhances the effect of mod(mdg4)ul on the y2 phenotype.

Suppresses the transformation of A6 to A7 seen in Abd-BFab7-P18.1 heterozygotes, but does not alter the phenotype of Abd-BFab7-12 heterozygotes.

Snr1 mutations enhance the abdominal transformations.

Suppresses the extra sex comb phenotype of Pc4, Ts(YLt;2Lt)L124. Causes between 50% and 100% suppression of the Pc4/+ extra sex combs phenotype.

Xenogenetic Interactions
Statement
Reference

The formation of ectopic wing veins observed in adult flies expressing Hsap\SMARCAL1Scer\UAS.cBa under the control of Scer\GAL4Bx-MS1096 is not affected by combination with a single copy of trxE2.

Complementation and Rescue Data
Images (0)
Mutant
Wild-type
Stocks (5)
Notes on Origin
Discoverer

Kennison, Tamkun.

External Crossreferences and Linkouts ( 0 )
Synonyms and Secondary IDs (4)
References (72)