mutant phenotype is characterized by distal gaps in the longitudinal veins.
13.2% of heterozygous females have a truncated L5 wing vein.
flies exhibit truncated wing veins.
Homozygotes lack the distal portion of wing vein L3 and lack most of the L4 and L5 wing veins.
mutants show loss of partial L3, L4 and L5 and weakened L2-L4 M veins, whereas heterozygous rhove-1
exhibit only a weakened distal portion of L5 vein.
flies show a partial loss of wing veins L3, L4 and L5.
The initial development of most veins in the wing is disrupted in rhove-1
mutants are viable, exhibiting partial loss of L3-L5 wing veins.
Homozygotes show incomplete wing veins L2, L3, L4 and L5.
Mutant embryos show duplication of the segmental border muscle and a complete loss of lateral adult muscle precursors.
mutant flies show loss of the distal ends of wing veins, particularly L3, L4 and L5.
The interactions between rhove-1
are additive. The interactions between rhove-1
are additive. emc1
clones in rhove-1 vn1
wings differentiate as veins in positions possibly corresponding to those of wild-type veins. Vein differentiation in these clones frequently fails in distal regions of veins.
In a e1
double mutant, in which all of the longitudinal veins are truncated or eliminated, ectopic melanin only develops in proximal areas that truncate before reaching the margin.
Germline clones fail to rescue the female sterile phenotype of Fs(3)Apc
Homozygotes lack the distal ends of all longitudinal wing veins.
Wings lack the longitudinal veins.
supresses the vein defects of bs
mutations although L2 to L5 remain truncated distally.
Wings have shortened longitudinal veins that do not reach the wing margin.
Lack distal segments of wing veins especially veins L4 and L5. Homozygous phenotype is strongly suppressed by heterozygous rhoWk
Wing veins do not reach margins.