mutants do not develop a wing pouch.
Mature mutant wing discs lack the wing primordium.
Homozygous clones induced in mid- to late second instar larvae are able to contribute normally to the wing pouch. However, homozygous clones induced at earlier times show a progressive decrease in their ability to do so. Homozygous clones induced during the first larval instar and given a competitive growth advantage using the Minute technique appear to be excluded from the wing pouch, forming non-wing tissue at its expense.
Mutants show no growth of wing blade tissue, although some hinge structures are still present.
Homozygotes lack halteres and wings are almost always absent.
Homozygous flies have virtually no wings.
Mutant adults show an almost complete lack of wings.
adults show an almost complete lack of wing structures.
Significant cell death is not seen in the third larval instar wing disc (similar to wild type).
Wing phenotype is unaffected by temperature.
Flies have knobbed wings and halteres.
Homozygotes display high rate of resistance to fluorodeoxyuridine (FUdR), an inhibitor of thymidylate synthetase and slight resistance to aminopterin.
Wing and haltere structures eliminated: cell death of larval imaginal discs.
Homozygotes show strong wing and haltere reductions. They are wild type with respect to the postscutellar bristle and female fertility phenotypes. Transheterozygotes with vg1
exhibit wing bubbles and unequal wing deficiencies. The complementation behaviour is unlikely to be mediated by a transvection-like process.
Homozygous viable, but with an extreme vg phenotype.
Homozygotes have a vgnw
wing phenotype. Postscutellars are wild-type, and halteres are missing.
homozygous wing phenotype like that of vgnw
halteres absent postscutellar bristles present homozygous viable female-fertile?