The co-expression of abd-AScer\UAS.cGa and rhoScer\UAS.cUa under the control of Scer\GAL4prd.PU enhances the increased and/or ectopic oenocytes in Prd-positive segments during stage 15 of embryogenesis displayed in either single expression condition.
Embryos coexpressing abd-AScer\UAS.cGa and Abd-BScer\UAS.cCa under the control of Scer\GAL4twi.PB have ectopic male-specific somatic gonadal precursor (msSGP) cells and ectopic somatic gonadal precursor (SGP) cells. A large group of SGPs and msSGPs associates with most of the germ cells in the region where the gonad would normally be coalescing and ectopic SGPs and msSGPs also associate with one another in more anterior regions.
Expression of abd-AScer\UAS.cGa under the control of Scer\GAL4lab.PH rescues the tritocerebral defects seen in lab14 embryonic brains. 28.4% of embryos show a complete rescue of the defects (taking into account that the phenotypic penetrance of the lab14 phenotype is 88.6%).
Scer\GAL4twi.PG induced expression of Abd-BScer\UAS.cCa blocks formation of the gonadal primordium, coexpression of Abd-BScer\UAS.cCa and abd-AScer\UAS.cGa restores gonadal mesoderm. Df(3R)Ubx109/Df(3R)P9 embryos and Df(3R)P9 homozygotes lack gonads, ectopic expression of abd-A restores the formation of an encapsulated gonad.
The spiracle phenotype of Df(3R)Ubx109 is modified in the abd-AScer\UAS.cGa, Scer\GAL4Kr.PM combination to suppress spiracle formation, irreversibly, wherever the ectopic protein is produced, i.e. completely in parasegments 7 and 8 and incompletely in parasegments 5,6 and 9. The phenotype of Df(3R)Ubx109 is modified in the abd-AScer\UAS.cGa, Scer\GAL4h-1J3 combination to suppress anterior spiracles in alternate parasegments. In the abd-AScer\UAS.cGa, Scer\GAL469B anterior spiracles are suppressed in all segments. The sensory organ phenotype of Df(3R)Ubx109 is modified in the abd-AScer\UAS.cGa, Scer\GAL4h-1J3 or abd-AScer\UAS.cGa, Scer\GAL4Kr.PM combination to prevent the ventral migration of the cells that would form the kolbchen, though later differentiation produces misplaced kolbchen rather than dorsal hairs. The sensory organ phenotype of Df(3R)Ubx109 is modified in the abd-AScer\UAS.cGa, Scer\GAL469B combination to affect the migration and differentiation of the kolbchen.