GT to AT mutation in the splice donor site
Mutation of the donor splice site of the third intron.
meiotic cell cycle defective | recessive
Meiotic recombination is reduced but not absent. Mutation does not interfere with ord4 activity: does not show negative complementation.
Transmission rate of Dp(1;f)J21A through females to progeny is 28%, ord mutation has no effect on transmission. Mutation has no effect on Dp(1;f)J21A transmission in males.
Level of chromosome missegregation in homozygous females is 60.2%.
Increased frequency of premature sister chromatid disjunction: homozygous females are fertile.
ord1/ord3 has phenotype, non-suppressible by anon-59Da3.6RR
ord1/ord3 is rescued by ordEGFP
ord1/ord3 is rescued by ord+t6.3
ord1/ord3 is rescued by ord+tD39
ord1/ord3 is not rescued by ordD26
ord1/ord3 is not rescued by ord7.3BPΔ
Does not interact with ord4 to alter the female meiotic chromosome segregation phenotype.