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General Information
Symbol
Dmel\rprGMR.PW
Species
D. melanogaster
Name
glass multimer reporter construct of White
FlyBase ID
FBal0050351
Feature type
allele
Associated gene
Associated Insertion(s)
Carried in Construct
Also Known As
GMR-rpr, GMR-reaper, PGMR-reaper
Allele class
Nature of the Allele
Allele class
Mutations Mapped to the Genome
 
Type
Location
Additional Notes
References
Associated Sequence Data
DNA sequence
Protein sequence
 
 
Progenitor genotype
Carried in construct
Cytology
Nature of the lesion
Statement
Reference

GMR regulatory sequences drive expression of a rpr cDNA.

Allele components
Product class / Tool use(s)
Encoded product / tool
Expression Data
Reporter Expression
Additional Information
Statement
Reference
 
Marker for
Reflects expression of
Reporter construct used in assay
Human Disease Associations
Disease Ontology (DO) Annotations
Models Based on Experimental Evidence ( 0 )
Disease
Evidence
References
Modifiers Based on Experimental Evidence ( 0 )
Disease
Interaction
References
Comments on Models/Modifiers Based on Experimental Evidence ( 0 )
 
Phenotypic Data
Phenotypic Class
Phenotype Manifest In
Detailed Description
Statement
Reference

rprGMR.PW flies have an eye ablation phenotype and show signs of cell death in the eye disc.

Eyes are highly ablated and abnormally shaped.

Expression of rprGMR.PW in the eye causes apoptosis in the developing retina that results in a small-eye phenotype.

Flies expressing rprGMR.PW have a rough eye phenotype.

rprGMR.PW mutants results in increased photoreceptor apoptotic cell death.

Adults expressing rprGMR.PW show a reduction in eye size compared to wild type.

rprGMR.PW flies show an eye ablation phenotype.

Expression of rprGMR.PW in the eye reduces the overall size and pigmentation of the eye.

Mosaic animals in which the eyes are expressing rprGMR.PW have an small eye phenotype.

Flies carrying rprGMR.PW have rough and reduced eyes.

Flies expressing rprGMR.PW have small, rough eyes.

Flies expressing rprGMR.PW have a small eye phenotype.

Expression of rprGMR.PW results in widespread ectopic apoptosis in the developing eye, resulting in mis-patterning of the ommatidial rows and a reduction in size of the adult eye field.

rprGMR.PW results in a dosage dependent eye degeneration phenotype.

rprGMR.PW flies have very small, rough eyes.

Flies expressing rprGMR.PW show an eye ablation phenotype.

flies exhibit moderate loss of eye tissue.

Flies expressing rprGMR.PW have a severely ablated eye.

Causes dose-dependent elimination of the eye due to ectopic apotosis. Eye is small and roughened.

Mutants have eyes that are severely reduced in size. The addition of BacA\p35GMR.PH to rprGMR.PW flies, completely suppresses the eye phenotype seen in those flies. The addition of Df(3L)AC1 to rprGMR.PW flies shows a significant suppression of the eye phenotype seen in these eyes.

Flies carrying rprGMR.PW have an ablated eye phenotype.

rprGMR.PW flies have a mild but easily detectable eye phenotype.

Flies carrying rprGMR.PW have a small eye phenotype and show increased numbers of dying cells posterior to the morphogenetic furrow.

Wild-type larvae reduce their path lengths show increased head swinging when exposed to light. These responses are not altered in larvae carrying rprGMR.PW. Wild-type larvae show a greater change in direction when lights are turned on or off (light (L) to dark (D), or D to L transition) than in the absence of a light transition (D to D). The amplitude of change of direction is greater for the D to L than for the L to D transition. The difference in the amplitude of change of direction at all transitions is abolished in larvae carrying rprGMR.PW.

Two copies of rprGMR.PW in an otherwise wild-type background result in a severely roughened eye.

Causes a dose-dependent mild eye ablation phenotype.

Flies carrying two copies of rprGMR.PW have noticeably roughened eyes with many fused or small ommatidia and extra bristles.

Eye is ablated due to apoptosis in a dose-dependent manner.

The eye is ablated, in a dose-dependent manner. Two doses of P{GMR-rpr.W} causes a rough eye, three doses decreases the size of the eye, four doses eliminates the eye completely. Examination of developing eye discs reveals that increased cell death causes the loss of the eye.

External Data
Interactions
Show genetic interaction network for Enhancers & Suppressors
Phenotypic Class
Enhanced by
Statement
Reference

rprGMR.PW has visible phenotype, enhanceable by DriceC1/Drice[+]

rprGMR.PW has increased cell death | larval stage phenotype, enhanceable by DriceC1/Drice[+]

rprGMR.PW has visible phenotype, enhanceable by Drice[+]/DriceC2

rprGMR.PW has increased cell death | larval stage phenotype, enhanceable by Drice[+]/DriceC2

rprGMR.PW has visible phenotype, enhanceable by PSR[+]/PSRFM1

rprGMR.PW has visible phenotype, enhanceable by PSRFM1/PSRFM1

rprGMR.PW has visible phenotype, enhanceable by Df(2R)017/+

rprGMR.PW has visible phenotype, enhanceable by Df(2R)PC66/+

rprGMR.PW has visible phenotype, enhanceable by Diap133-1s

rprGMR.PW has visible phenotype, enhanceable by Diap121-4s

NOT Enhanced by
Statement
Reference
Suppressed by
Statement
Reference

rprGMR.PW has visible phenotype, suppressible by DriceΔ1/Drice17

rprGMR.PW has visible phenotype, suppressible by DriceΔ1/DriceL1

rprGMR.PW has visible phenotype, suppressible by DriceΔ1/DriceL2

rprGMR.PW has visible phenotype, suppressible by DriceC1/DriceΔ1

rprGMR.PW has visible phenotype, suppressible by DriceΔ1/DriceC2

rprGMR.PW has visible phenotype, suppressible by DriceΔ1/DriceS1

rprGMR.PW has visible phenotype, suppressible by DriceΔ1/DriceS2

rprGMR.PW has visible phenotype, suppressible | partially by Drice[+]/DriceL1

rprGMR.PW has increased cell death | larval stage phenotype, suppressible | partially by Drice[+]/DriceL1

rprGMR.PW has visible phenotype, suppressible | partially by Drice[+]/DriceL2

rprGMR.PW has visible phenotype, suppressible | partially by DriceS1/Drice[+]

rprGMR.PW has visible phenotype, suppressible | partially by Drice17/Drice17

rprGMR.PW has visible phenotype, suppressible by pnut[+]/pnutXP

rprGMR.PW has visible phenotype, suppressible by pnut[+]/pnut1

rprGMR.PW has visible phenotype, suppressible | partially by klu[+]/kluunspecified

rprGMR.PW has visible phenotype, suppressible by morgue[+]/morgueEP1184/Scer\GAL4[-]

rprGMR.PW has increased cell death phenotype, suppressible by morgue[+]/morgueEP1184/Scer\GAL4[-]

rprGMR.PW has visible phenotype, suppressible | partially by eff598/eff[+]

rprGMR.PW has visible phenotype, suppressible | partially by eff[+]/effD73

rprGMR.PW has visible phenotype, suppressible by Df(2L)morgue19/+

rprGMR.PW has visible phenotype, suppressible | partially by Df(3L)AC1

rprGMR.PW has visible phenotype, suppressible by clnSF3-2

rprGMR.PW has visible phenotype, suppressible by Darkk11502

rprGMR.PW has visible phenotype, suppressible by DarkCD4

rprGMR.PW has visible phenotype, suppressible by Df(1)su(s)R194

NOT suppressed by
Statement
Reference

rprGMR.PW has visible phenotype, non-suppressible by DriceΔ1/Drice[+]

rprGMR.PW has visible phenotype, non-suppressible by Drice[+]/Drice17

rprGMR.PW has visible phenotype, non-suppressible by Darkunspecified/Ark[+]

rprGMR.PW has visible phenotype, non-suppressible by TER94[+]/TER9426-8

rprGMR.PW has visible phenotype, non-suppressible by DammC156G.GMR.Tag:FLAG

Enhancer of
Statement
Reference

rprGMR.PW is an enhancer of visible phenotype of Dcp-1fl.GMR

rprGMR.PW is an enhancer of visible phenotype of Dricefl.GMR

Other
Statement
Reference
Phenotype Manifest In
Enhanced by
Statement
Reference

rprGMR.PW has eye phenotype, enhanceable by DriceC1/Drice[+]

rprGMR.PW has eye disc | larval stage phenotype, enhanceable by DriceC1/Drice[+]

rprGMR.PW has eye phenotype, enhanceable by Drice[+]/DriceC2

rprGMR.PW has eye disc | larval stage phenotype, enhanceable by Drice[+]/DriceC2

rprGMR.PW has eye phenotype, enhanceable by PSR[+]/PSRFM1

rprGMR.PW has eye phenotype, enhanceable by PSRFM1/PSRFM1

rprGMR.PW has eye phenotype, enhanceable by Df(2R)017/+

rprGMR.PW has eye phenotype, enhanceable by Df(2R)PC66/+

rprGMR.PW has phenotype, enhanceable by Bruceunspecified/Bruce[+]

rprGMR.PW has phenotype, enhanceable by Df(3R)faf-BP/+

rprGMR.PW has phenotype, enhanceable by Pros26[+]/Prosβ61

rprGMR.PW has eye phenotype, enhanceable by Diap133-1s

rprGMR.PW has eye phenotype, enhanceable by Diap121-4s

rprGMR.PW has phenotype, enhanceable by Diap14

rprGMR.PW has phenotype, enhanceable by Diap15

rprGMR.PW has phenotype, enhanceable by Diap111-3e

rprGMR.PW has eye phenotype, enhanceable by Diap1109.07

rprGMR.PW has eye phenotype, enhanceable by Diap14

rprGMR.PW has eye phenotype, enhanceable by Diap15

rprGMR.PW has eye phenotype, enhanceable by Diap16B

rprGMR.PW has eye phenotype, enhanceable by Diap17

rprGMR.PW has eye phenotype, enhanceable by Diap181.03

rprGMR.PW has eye phenotype, enhanceable by Diap19

rprGMR.PW has eye phenotype, enhanceable by Dcp-1GMR.PS

rprGMR.PW has eye phenotype, enhanceable by Ras85De2F

rprGMR.PW has eye phenotype, enhanceable by Raf7

NOT Enhanced by
Statement
Reference

rprGMR.PW has phenotype, non-enhanceable by Ubc2k13206

rprGMR.PW has phenotype, non-enhanceable by lwrunspecified

rprGMR.PW has phenotype, non-enhanceable by SM2-1SM2-1

rprGMR.PW has phenotype, non-enhanceable by SM3-3SM3-3

rprGMR.PW has phenotype, non-enhanceable by SM3-6SM3-6

rprGMR.PW has phenotype, non-enhanceable by SM3-8SM3-8

rprGMR.PW has phenotype, non-enhanceable by bulSF3-1

rprGMR.PW has phenotype, non-enhanceable by stySM3-5

rprGMR.PW has phenotype, non-enhanceable by stySM3-9

rprGMR.PW has phenotype, non-enhanceable by osaE65

rprGMR.PW has eye phenotype, non-enhanceable by Ras85DN17.sev

rprGMR.PW has eye phenotype, non-enhanceable by Ras85DV12.sev

Suppressed by
Statement
Reference

rprGMR.PW has eye phenotype, suppressible by DriceΔ1/Drice17

rprGMR.PW has eye phenotype, suppressible by DriceΔ1/DriceL1

rprGMR.PW has eye phenotype, suppressible by DriceΔ1/DriceL2

rprGMR.PW has eye phenotype, suppressible by DriceC1/DriceΔ1

rprGMR.PW has eye phenotype, suppressible by DriceΔ1/DriceC2

rprGMR.PW has eye phenotype, suppressible by DriceΔ1/DriceS1

rprGMR.PW has eye phenotype, suppressible by DriceΔ1/DriceS2

rprGMR.PW has eye phenotype, suppressible | partially by Drice[+]/DriceL1

rprGMR.PW has eye disc | larval stage phenotype, suppressible | partially by Drice[+]/DriceL1

rprGMR.PW has eye phenotype, suppressible | partially by Drice[+]/DriceL2

rprGMR.PW has eye phenotype, suppressible | partially by DriceS1/Drice[+]

rprGMR.PW has eye phenotype, suppressible by Cdk7S164A.T170A

rprGMR.PW has eye phenotype, suppressible by Diap1αTub84B.WT

rprGMR.PW has eye phenotype, suppressible | partially by Drice17/Drice17

rprGMR.PW has eye phenotype, suppressible | partially by lzts1

rprGMR.PW has eye | somatic clone phenotype, suppressible by DroncL32/DroncL32

rprGMR.PW has eye | somatic clone phenotype, suppressible by DroncI24/DroncI24

rprGMR.PW has eye phenotype, suppressible by pnut[+]/pnutXP

rprGMR.PW has eye phenotype, suppressible by pnut[+]/pnut1

rprGMR.PW has ommatidium phenotype, suppressible | partially by klu[+]/kluunspecified

rprGMR.PW has eye phenotype, suppressible by morgue[+]/morgueEP1184/Scer\GAL4[-]

rprGMR.PW has ommatidium phenotype, suppressible by morgue[+]/morgueEP1184/Scer\GAL4[-]

rprGMR.PW has eye phenotype, suppressible | partially by Df(2L)M24F-B/+

rprGMR.PW has eye phenotype, suppressible | partially by Df(2L)dp-h25/+

rprGMR.PW has eye phenotype, suppressible | partially by Df(2L)ed-dp/+

rprGMR.PW has ommatidium phenotype, suppressible | partially by Df(2L)ed-dp/+

rprGMR.PW has eye phenotype, suppressible | partially by Df(3R)H-B79/+

rprGMR.PW has ommatidium phenotype, suppressible | partially by Df(3R)H-B79/+

rprGMR.PW has eye phenotype, suppressible | partially by bsk2/bsk[+]

rprGMR.PW has ommatidium phenotype, suppressible | partially by bsk2/bsk[+]

rprGMR.PW has eye phenotype, suppressible | partially by hep[+]/hep1

rprGMR.PW has ommatidium phenotype, suppressible | partially by hep[+]/hep1

rprGMR.PW has eye phenotype, suppressible | partially by Df(2L)sc19-8/+

rprGMR.PW has eye phenotype, suppressible | partially by eff598/eff[+]

rprGMR.PW has eye phenotype, suppressible | partially by eff[+]/effD73

rprGMR.PW has eye phenotype, suppressible by Df(2L)morgue19/+

rprGMR.PW has phenotype, suppressible by Diap121-2s

rprGMR.PW has phenotype, suppressible by Diap123-4s

rprGMR.PW has phenotype, suppressible by Diap123-8s

rprGMR.PW has phenotype, suppressible by Diap145-2s

rprGMR.PW has phenotype, suppressible by Diap16-3s

rprGMR.PW has eye phenotype, suppressible by Diap1SL

rprGMR.PW has phenotype, suppressible | partially by Df(3L)AC1

rprGMR.PW has phenotype, suppressible by BacA\p35GMR.PH

rprGMR.PW has eye phenotype, suppressible | partially by Df(3L)AC1

rprGMR.PW has eye phenotype, suppressible by clnSF3-2

rprGMR.PW has eye phenotype, suppressible by Darkk11502

rprGMR.PW has ommatidium phenotype, suppressible by DarkCD4

rprGMR.PW has eye phenotype, suppressible by DarkCD4

rprGMR.PW has eye phenotype, suppressible by Df(1)su(s)R194

rprGMR.PW has ommatidium phenotype, suppressible by Df(1)su(s)R194

rprGMR.PW has eye phenotype, suppressible by Ras85DV12.sev

rprGMR.PW has phenotype, suppressible by Df(3L)X14/+

rprGMR.PW has eye phenotype, suppressible by Dsor1Su1

rprGMR.PW has eye phenotype, suppressible by rlSem

NOT suppressed by
Statement
Reference

rprGMR.PW has eye phenotype, non-suppressible by DriceΔ1/Drice[+]

rprGMR.PW has eye phenotype, non-suppressible by Drice[+]/Drice17

rprGMR.PW has eye phenotype, non-suppressible by Diap1D20A.αTub84B

rprGMR.PW has eye phenotype, non-suppressible by Diap1N21M.21-438.αTub84B

rprGMR.PW has eye phenotype, non-suppressible by Diap1C406Y.αTub84B

rprGMR.PW has eye phenotype, non-suppressible by Darkunspecified/Ark[+]

rprGMR.PW has eye phenotype, non-suppressible by TER94[+]/TER9426-8

rprGMR.PW has phenotype, non-suppressible by lwrunspecified

rprGMR.PW has phenotype, non-suppressible by Ubc2k13206

rprGMR.PW has eye phenotype, non-suppressible by DammC156G.GMR.Tag:FLAG

rprGMR.PW has phenotype, non-suppressible by SM2-1SM2-1

rprGMR.PW has phenotype, non-suppressible by SM3-3SM3-3

rprGMR.PW has phenotype, non-suppressible by SM3-6SM3-6

rprGMR.PW has phenotype, non-suppressible by SM3-8SM3-8

rprGMR.PW has phenotype, non-suppressible by bulSF3-1

rprGMR.PW has phenotype, non-suppressible by stySM3-5

rprGMR.PW has phenotype, non-suppressible by stySM3-9

rprGMR.PW has eye phenotype, non-suppressible by Darkk11502

rprGMR.PW has eye phenotype, non-suppressible by Ras85DN17.sev

rprGMR.PW has eye phenotype, non-suppressible by Ras85DV12.sev

Enhancer of
Statement
Reference

rprGMR.PW is an enhancer of eye phenotype of Dcp-1fl.GMR

rprGMR.PW is an enhancer of eye phenotype of Dricefl.GMR

rprGMR.PW is an enhancer of eye phenotype of Dcp-1GMR.PS

Other
Statement
Reference
Additional Comments
Genetic Interactions
Statement
Reference

Drice17/DriceΔ1, DriceΔ1/DriceL1, DriceΔ1/DriceL2, DriceΔ1/DriceC1, DriceΔ1/DriceC2, DriceΔ1/DriceS1, DriceΔ1/DriceS2 (but not DriceΔ1/+) significantly suppresses the eye ablation phenotype in rprGMR.PW flies.

DriceC1/+ or DriceC2/+ significantly enhance and DriceL1/+, DriceL2/+, DriceS1/+ or DriceS2/+ weakly suppress the eye ablation phenotype in rprGMR.PW flies; Drice17/+ does not suppress the phenotype. DriceL1/+ significantly suppresses and DriceC1/+ or DriceC2/+ significantly enhance the increased cell death seen in rprGMR.PW larval eye discs.

Expression of CG3251GD10916 under the control of Scer\GAL4GMR.PF enhances the rprGMR.PW-induced cell death in the eye.

Expression of CG3251KK106465 under the control of Scer\GAL4GMR.PF enhances the rprGMR.PW-induced cell death in the eye.

A Cdk7S164A.T170A mutant background significantly suppresses the partial eye ablation found upon expression of rprGMR.PW in the developing eye.

Ras85DR68Q very mildly suppresses rprGMR.PW-induced cell death although this is suspected to be due to suppression of endogenous W activity and not grim or rpr activity.

Scer\GAL4GMR.PF-mediated expression of P{Sym-UAS-Hsrω} significantly rescues the rprGMR.PW eye phenotype.

Scer\GAL4GMR.PF-mediated expression of HsrωEP3037 significantly enhances the rprGMR.PW eye phenotype.

Scer\GAL4GMR.PF-mediated expression of P{Sym-UAS-Hsrω} eliminates the enhancing effect of HsrωEP3037 on rprGMR.PW eyes as well as reversing the level of degeneration seen in eyes expressing rprGMR.PW alone.

Expression of thScer\UAS.cHa using Scer\GAL4GMR.PF rescues the rprGMR.PW small eye phenotype.

Co-expression of P{Sym-UAS-Hsrω} in the rprGMR.PW, thScer\UAS.cHa, Scer\GAL4GMR.PF background improves eye morphology such that eyes are indistinguishable from wild type.

Expression of thdsRNA.Scer\UAS.cLa using Scer\GAL4GMR.PF enhances the rprGMR.PW small eye phenotype.

Co-expression of P{Sym-UAS-Hsrω} fails to suppress the eye damage in the rprGMR.PW, thdsRNA.Scer\UAS.cLa, Scer\GAL4GMR.PF background.

Expression of thαTub84B.WT or thN.21-438.αTub84B suppresses the small-eye phenotype observed in rprGMR.PW mutants.

Expression of thD20A.αTub84B or thN21M.21-438.αTub84B fails to suppress the small-eye phenotype observed in rprGMR.PW mutants.

Expression of thC406Y.αTub84B, which lacks a functional RING domain, fails to inhibit the small-eye phenotype observed in rprGMR.PW mutants.

The rough eye phenotype caused by rprGMR.PW is enhanced by one copy of PSRFM1 and is further enhanced by two copies of PSRFM1.

Expression of scramb1Scer\UAS.cAa, scramb2Scer\UAS.cAa, or both, under the control of Scer\GAL4Act does not affect rprGMR.PW-induced photoreceptor apoptosis. The eye sizes are not significantly different in the three mutant backgrounds compared with control flies.

CskIR.Scer\UAS; Scer\GAL4GMR.PF partially suppresses the reduction in eye size due to rprGMR.PW.

The eye ablation phenotype caused by rprGMR.PW is weakly suppressed by homozygosity for Ice17.

Reducing lz activity by transferring lzts1 rprGMR.PW flies to the non-permissive temperature of 33oC partially suppresses the rprGMR.PW eye phenotype.

The severity of the rprGMR.PW eye phenotype is suppressed by "dronc[d5]" animals (Df(3L)NcZ in which CG6685 function, but not Nc function, is rescued by P{CG6685+tDa}).

The reduced eye phenotype caused by expression of rprGMR.PW is significantly suppressed by pnut1/+ or pnutXP/+, but Arkunspecified/+ has little effect.

The eye phenotype caused by rprGMR.PW is mildly but consistently suppressed by kluunspecified/+.

The rprGMR.PW eye phenotypes are significantly suppressed by heterozygosity for Df(2L)sc19-8, Df(2L)ed-dp, Df(2L)dp-h25, Df(2L)M24F-B, Traf1EP578, Df(3R)H-B79, bsk2 or hep1, but not Df(2L)tkv3 or Df(2L)ed1.

The eye ablation phenotype of rprGMR.PW flies is partially suppressed by effD73/+.

The small eye phenotype of rprGMR.PW flies is significantly enhanced by th21-4s or th33-1s.

The addition of sklScer\UAS.cWa (driven by Scer\GAL4GMR.PF) to rprGMR.PW flies enhances the eye phenotype. Flies exhibit much more severe eye cell death, with greatly reduced size and pigmentation. The subsequent addition of BacA\p35Scer\UAS.cHa represses these phenotypes.

The eye cell death phenotype caused by rprGMR.PW is suppressed by Df(2L)morgue19/+.

The rprGMR.PW severely ablated eye phenotype is not suppressed by DammC156G.GMR.T:Zzzz\FLAG.

The addition of rprGMR.PW to Dcp-1fl.GMR flies show a strong enhancement of the eye phenotype seen in Dcp-1fl.GMR flies. The addition of rprGMR.PW to Icefl.GMR flies show a strong enhancement of the eye phenotype seen in Icefl.GMR flies.

The eye phenotype caused by expression of rprGMR.PW is significantly suppressed by Arkk11502.

The rprGMR.PW rough eye phenotype is strongly suppressed if the flies are also homozygous for ArkCD4.

Arkk11502 has little effect on the eye phenotype caused by rprGMR.PW. A strong synergism in cell ablation is seen when Dcp-1GMR.PS and rprGMR.PW are coexpressed in the eye. This phenotype is not modified by Arkk11502.

The grimGMR.PC eye ablation phenotype is not particularly affected by either Ras85DV12.sev and Ras85DN17.sev.

The rprGMR.PW rough eye phenotype is suppressed if the flies are also heterozygous for Df(1)su(s)R194. The rough eye phenotype is restored in flies carrying rprGMR.PW, heterozygous for Df(1)su(s)R194 and also carrying Dredd+tCa.

Loss of function alleles of Egfr and spi dominantly enhance the rprGMR.PW mutant phenotype. Reduction in W dosage also suppresses the rprGMR.PW mutant phenotype.

Expression of BacA\p35 abrogates the effects of ectopic rpr; flies carrying both P{GMRP35} and P{GMR-rpr.W} (two copies) show the same slightly rough eye phenotype as flies carrying P{GMRP35} alone.

Xenogenetic Interactions
Statement
Reference
Complementation and Rescue Data
Comments
Images (0)
Mutant
Wild-type
Stocks (0)
Notes on Origin
Discoverer
External Crossreferences and Linkouts ( 0 )
Synonyms and Secondary IDs (2)
Reported As
Symbol Synonym
rprGMR.PW
Name Synonyms
glass multimer reporter construct of White
Secondary FlyBase IDs
    References (50)