adult thorax | dorsal & chaeta | somatic clone
mechanosensory chaeta & adult abdomen | somatic clone
mechanosensory chaeta | supernumerary & adult abdomen | somatic clone
socket & adult abdomen | somatic clone
The loss of wing veins seen in animals expressing brmK804R.Scer\UAS.T:Ivir\HA1 under the control of Scer\GAL469B is suppressed by Snr1R3. The Dl9P thick vein phenotype is enhanced by Snr1E1/Snr1R3. dpps1/dpps1 Snr1E1/Snr1R3 animals show suppression of the Snr1 extra wing vein phenotype and enhancement of the dpp loss of vein phenotype in L5. Snr1E1/Snr1R3 suppresses the loss of wing vein L5 seen in rhove-1 homozygotes and rhove-1 suppresses the Snr1E1/Snr1R3 extra wing vein phenotype.
Rpd304556/Snr1R3 double heterozygotes have wing vein defects; 32% have normal wings, 38% have an ectopic vein emanating from the posterior crossvein and 30% have ectopic veins emanating from the posterior crossvein and posterior to the L5 longitudinal vein. The ectopic wing vein phenotype seen in Rpd304556/Snr1R3 double heterozygotes at 29oC is partially suppressed by brm2/+.
Homozygous Snr1R3 clones induced in a brm2/+ background are less frequently observed and are smaller in size compared to both brm+ and control clones induced at similar stages, suggesting an additive effect between brm and Snr1. The presence of brm2/+ enhances the Snr1R3 clone phenotype in the abdomen. The AntpNs/+ antenna to leg transformation is suppressed about 50% by Snr1R3.