|Feature type||allele||Associated gene||Dmel\ed|
|Also Known As||l(2)k01102, k01102|
|Map ( GBrowse )||
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|Nature of the Allele|
|Mutations Mapped to the Genome|
|Associated Sequence Data|
|Nature of the lesion|
|Caused by insertion|
|Phenotype Manifest In|
Heterozygotes show a direct effect on the shape of the wing.
edk01102/edm1 animals usually die as pupae, but adult escapers are sometimes produced in low density cultures. These escapers have mild defects in the leg; the legs have the correct number of segments, but the sex combs are malformed in males. They also have a rough eye phenotype.
edlH23/edk01102 flies have extra dorsocentral, scutellar and recurved wing margin bristles (chemosensory ventral triple row + dorsal triple row). ed6.1/edk01102 and edk01102/Df(2L)ed-dp flies have extra dorsocentral and scutellar bristles.
Homozygotes are viable until the late third larval instar. Homozygous and edk01102/edslA12 eye discs contain ommatidia with multiple R8 cells. This phenotype is also seen in homozygous clones in mosaic eye discs. Ommatidia in homozygous clones in the eye sometimes contain extra R7 cells and in all cases where ommatidia contain extra R7 cells, extra R8 cells are also present.
|Phenotype Manifest In|
edk01102/+ strongly enhances the EgfrE1/+ phenotype, resulting in eyes that are approximately 1/3 the size of those of EgfrE1/+ flies. argosΔ7/+ mildly enhances the edk01102 phenotype; the number of ommatidia with multiple R8 cells is increased from 18% to 34% in the double mutant flies. argosW11 enhances the edk01102 phenotype; the number of ommatidia with multiple R8 cells is increased from 21% to 46% in the double mutant flies.
|Complementation & Rescue Data|
|Fails to complement|
|Stocks ( 2 )|
|Notes on Origin|
Separable from: a second site lethal mutation on the second chromosome.
|External Crossreferences & Linkouts|
|Synonyms & Secondary IDs ( 5 )|
|Secondary FlyBase IDs|
|References ( 14 )|
|Personal communication to FlyBase|