The expression of N^int.G.UAS under the control of Scer\GAL4^pnt-14-94 results in larval brain type II neuroblasts with clustered mitochondria to one side of the nucleus.

The expression of N^{int.G.Scer\UAS} under the control of Scer\GAL4^sca-C253 leads to the virtual loss of thoracic sensory macrochaeta and microchaeta in adults, as compared to controls.

Under control conditions, the adulthood-only expression of N^int.G.UAS under the control of Scer\GAL4^esg-NP7397 (and Gal80[ts], for the temporal regulation of expression) leads to a significant decrease in the number of eggs laid by females, as compared to controls.

Expression of N^{int.G.Scer\UAS} under the control of Scer\GAL4^ptc.PU results in the production of cuticle folds along the proximo-distal axis of the leg that resemble ectopic joints.

N^{int.G.Scer\UAS} clones induced in type II neuroblasts (NBs) under the control of Scer\GAL4^αTub84B.PL contain ectopic NB-like cells (Dpn and Mira positive).

There is a reduction in the number of SELK neurons in animals expressing N^{int.G.Scer\UAS} under the control of Scer\GAL4^elav-C155.

Expression of N^{int.G.Scer\UAS} in glial cells, driven by Scer\GAL4^repo, leads to an irregular migration of peripheral glial cells. The dorsal intersegmental peripheral glial cells do not migrate to their normal ventral position.

Adults carrying N^{int.G.Scer\UAS}, Scer\GAL4^esg-NP5130 and Scer\GAL80^ts.αTub84B and raised at the permissive temperature have wild-type midguts (at this temperature, the Scer\GAL80^ts.αTub84B protein inhibits Scer\GAL4^esg-NP5130). When these flies are shifted to the non-permissive temperature (which inactivates Scer\GAL80^ts.αTub84B, allowing expression of N^{int.G.Scer\UAS} under the control of Scer\GAL4^esg-NP5130), some esg⁺ cells in the midgut show an increase in nuclear size.

Expression of N^{int.G.Scer\UAS} under the control of Scer\GAL4^GMR.PF produces excess mitoses in the eye disc.

Embryos expressing N^{int.G.Scer\UAS} under the control of Scer\GAL4^Kr.PM show an almost complete loss of neurons in parasegments 4-6.

Expression of N^{int.G.Scer\UAS} under the control of Scer\GAL4^sca-C253 removes essentially all bristles on the notum.

When N^{int.G.Scer\UAS} is driven by Scer\GAL4^eve.eme no DA1 muscles are formed and in 60% of hemisegments an increase in eve expressing pericardial cells (EPCs) is seen from two to three or four. An associated loss of DA1 and DO2 muscle precursors is seen.

N^{int.G.Scer\UAS} driven by Scer\GAL4^ap-md544 causes overproliferation in the wing disc.

In somatic clones where the expression of N^{int.G.Scer\UAS} is under the control of Scer\GAL4^Ubx.PdC various phenotypes are seen. Clones in the wing pouch of both surfaces cause wing outgrowths. The outgrowths contain small territories of mutant cells in their tip that differentiate wing margin pattern elements and wild-type cells in their base that differentiate wing blade tissue with identifiable vein patterns. The largest outgrowths appear in clones close to the anterior posterior boundary, away from the dorsal ventral boundary. Somatic clones where the expression of N^{int.G.Scer\UAS} is under the control of Scer\GAL4^Act5C.PI grow normally, without causing outgrowths.

When expression is driven by Scer\GAL4^btl.PS, dorsal and lateral truck fusion is prevented. Some dorsal branches extend an extra terminal branch. In 20% of dorsal branches cell markers suggest the presumptive fusion cell is converted to a terminal cell.

dMP2 neurons project their axons in an anterior direction, along the pathway of the vMP2, in embryos expressing N^{int.G.Scer\UAS} under the control of Scer\GAL4^ftz.ng.

Animals expressing N^{int.G.Scer\UAS} under the control of Scer\GAL4¹¹⁵¹ lack both dorsal longitudinal and dorsoventral muscles. The direct flight muscles (DFMs) appear relatively normal, although DFM 54 is thinner than normal.

When expression is driven by Scer\GAL4^ey.PH, the eye is enlarged, mainly in the dorso-ventral axis.

Embryos expressing N^{int.G.Scer\UAS} under the control of Scer\GAL4^twi.PG and Scer\GAL4^how-24B show no heart formation. The specification and/or differentiation of the visceral mesoderm and somatic muscles is also abolished.

Visceral mesoderm does not form in embryos expressing N^{int.G.Scer\UAS} under the control of both Scer\GAL4^how-24B and Scer\GAL4^twi.PG.

Expression of N^{int.G.Scer\UAS} under the control of Scer\GAL4^dpp.blk1 results in ectopic joint structures in the leg. The strength of the phenotype depends on the P{UAS-N.intra.G} line used. Weakly expressing lines develop normal size legs which develop partial ectopic joint structures, particularly in the tibia and first tarsal segment. The ectopic joint structures are restricted to the dorsal side of the leg. Flies that express N^{int.G.Scer\UAS} more strongly have legs with extremely abnormal morphology; the tibia and femur are bifurcated by an abnormal proximal-distal fold, and connections between tarsal segments are also affected, with many joint structures appearing in abnormal positions.

Embryos expressing N^{int.G.Scer\UAS} under the control of Scer\GAL4^C612b show an increase in the number of adult muscle precursors at the expense of the dorsal muscles.

When expression is driven by Scer\GAL4^Ubx.PdC in clones in the wing, outpocketings of the wing occur which include both N^{int.G.Scer\UAS}-expressing and neighboring wild type cells. Ectopic wing margin bristles appear in both mutant and wild type cells. The cells are smaller and have altered polarity.

N^int.G.UAS, Scer\GAL4^αTub84B.PL has abnormal neuroanatomy | somatic clone | third instar larval stage phenotype, suppressible by Su(H)^del47

N^int.G.UAS, Scer\GAL4^αTub84B.PL has increased cell number | somatic clone | third instar larval stage phenotype, suppressible by Su(H)^del47

N^int.G.UAS, Scer\GAL4^Kr.PM has abnormal neuroanatomy phenotype, suppressible by ed^ts/ed^2B8

N^int.G.UAS, Scer\GAL4^αTub84B.PL has abnormal neuroanatomy | somatic clone | third instar larval stage phenotype, non-suppressible by dpn⁷

N^int.G.UAS, Scer\GAL4^αTub84B.PL has increased cell number | somatic clone | third instar larval stage phenotype, non-suppressible by dpn⁷

N^int.G.UAS, Scer\GAL4^sca-C253 has visible phenotype, non-suppressible by ed^ΔECD.UAS, Scer\GAL4^sca-C253

N^int.G.UAS, Scer\GAL4^αTub84B.PL has type II neuroblast | ectopic | somatic clone | third instar larval stage phenotype, suppressible by Su(H)^del47

N^int.G.UAS, Scer\GAL4^Kr.PM has larval neuron phenotype, suppressible by ed^ts/ed^2B8

N^int.G.UAS, Scer\GAL4^eve.eme has muscle cell of mesothoracic dorsal acute muscle 1 phenotype, suppressible | partially by numb^{fl.UAS.Tag:MYC}, Scer\GAL4^eve.eme

N^int.G.UAS, Scer\GAL4^eve.eme has muscle cell of metathoracic dorsal acute muscle 1 phenotype, suppressible | partially by numb^{fl.UAS.Tag:MYC}, Scer\GAL4^eve.eme

N^int.G.UAS, Scer\GAL4^eve.eme has pericardial cell | ectopic phenotype, suppressible | partially by numb^{fl.UAS.Tag:MYC}, Scer\GAL4^eve.eme

N^int.G.UAS, Scer\GAL4^eve.eme has muscle cell of A1-7 dorsal acute muscle 1 phenotype, suppressible | partially by numb^{fl.UAS.Tag:MYC}, Scer\GAL4^eve.eme

N^int.G.UAS, Scer\GAL4^sca-C253 has macrochaeta | adult stage phenotype, non-suppressible by Rbfox1^RNAi.UAS, Scer\GAL4^sca-C253

N^int.G.UAS, Scer\GAL4^sca-C253 has microchaeta | adult stage phenotype, non-suppressible by Rbfox1^RNAi.UAS, Scer\GAL4^sca-C253

N^int.G.UAS, Scer\GAL4^ptc.PU has tarsal segment phenotype, non-suppressible by dysf³/dysf²

N^int.G.UAS, Scer\GAL4^ptc.PU has leg joint | ectopic phenotype, non-suppressible by dysf³/dysf²

N^int.G.UAS, Scer\GAL4^αTub84B.PL has type II neuroblast | ectopic | somatic clone | third instar larval stage phenotype, non-suppressible by dpn⁷

N^int.G.UAS, Scer\GAL4^sca-C253 has macrochaeta phenotype, non-suppressible by ed^ΔECD.UAS, Scer\GAL4^sca-C253

N^int.G.UAS, Scer\GAL4^bbg-C96 has wing sensillum phenotype, non-suppressible by mam^N.UAS, Scer\GAL4^bbg-C96

N^int.G.UAS/Scer\GAL4^ptc.PU is a non-suppressor of tarsal segment phenotype of dysf³/dysf²

N^int.G.UAS/Scer\GAL4^ptc.PU is a non-suppressor of leg joint phenotype of dysf³/dysf²

N^int.G.UAS/Scer\GAL4^ftz.ng is a non-suppressor of vMP2 neuron phenotype of mam^3.177