Embryos expressing dallyScer\UAS.cJa under the control of Scer\GAL4elav.PU show ISNb motor axon guidance defects (53.8% of hemisegments) and SNa motor axon guidance defects (25.5% of hemisegments).
Expression of dallyScer\UAS.cJa in the inner germarium sheath (IGS) cells under the control of Scer\GAL4C587 results in tumorous germaria that are filled with bam-negative GSC-like cells.
Expression of dallyScer\UAS.cJa under the control of Scer\GAL4en-e16E increases the posterior:anterior size ratio of the appendage-forming region of the haltere disc from the wild-type ratio of 0.48 to 0.59. The overall size of the adult haltere is 5% larger in animals of this genotype than in controls.
Expression of dallyScer\UAS.cJa in the wing, under the control of Scer\GAL4en-e16E, results in enlargement of the posterior portion by 46%, and enlargement of the anterior portion by a more modest 13%. In addition, the formation of the posterior and anterior crossveins are disrupted in these wings.
Expression of dallyScer\UAS.cJa in the eye, driven by Scer\GAL4ey.PH, causes an eye overgrowth phenotype. Ommatidia in these eyes are misshapen, with a reduced number of photoreceptors per ommatidial unit.
Expression of dallyScer\UAS.cJa under the control of Scer\GAL4A9 in a dallygem/dallyΔP-527 or dallygem mutant background results in the formation of excess dorsocentral bristles (the excess is around 30% compared to wild-type wings).
The posterior compartment/anterior compartment width ratio is increased in the haltere pouches of larvae expressing dallyScer\UAS.cJa under the control of Scer\GAL4en-e16E compared to control discs.