Homozygous clones generated at the early first larval instar in the peripodial epithelium of the wing disc survive.
Homozygous clones generated at the early first larval instar in the disc proper of the wing disc show reduced survival compared to wild-type twin spots. Those that do survive are much smaller than their respective wild-type twin spots.
Homozygous clones generated at the second larval instar in the disc proper of the wing disc survive poorly compared to their wild-type twin spots. The surviving clones extrude basally from the disc proper. Occasionally, clones that originate in the disc proper grow to a large size.
Homozygous clones generated at the mid- to late third larval instar in the disc proper and the posterior lateral margin of the hinge of the wing disc minimise contact with their wild-type neighbours are form round vesicles that extrude basally.
The 4o fate of the dorsal epidermis is replaced with 1o-3o fates in mutant embryos.
In linunspecified homozygous embryos, both the foregut and the hindgut are bloated, boundary cells are missing from the hindgut, and the keyhole region of the foregut is expanded.
The hindgut is wider and shorter than normal in mutant embryos. The hindgut appears distended, the large intestine is missing and the boundary cell rows that normally run along the length of the large intestine are missing. The foregut is shorter and wider than normal and the proventriculus does not form.
The cuticle phenotype of embryos lacking both maternal and zygotic lin activity is indistinguishable from that of zygotic lin mutants.
A small anterior portion of each segment is deleted in linunspecified mutant embryos. Segment A8, spiracles and anal plates are absent. The head is abnormal.