|Feature type||allele||Associated gene||Dmel\α-Cat|
|Mutagen||in vitro construct - regulatory fusion, in vitro construct - coding region fusion|
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Controlled Vocabulary Terms
|All updates||Click here to see a list of all updates to this record from FB2010_08 and on.|
|Nature of the Allele|
|Mutations Mapped to the Genome|
|Associated Sequence Data|
|Nature of the lesion|
|Carried in construct|
(Wang et al., 2006, Kaltschmidt et al., 2002, Oda and Tsukita, 1999, Le Borgne et al., 2002, Woolner et al., 2005, Roch et al., 2003, Simoes et al., 2006, Grieder et al., 2008, Larson et al., 2008, Schock and Perrimon, 2002, Knox and Brown, 2002, Wood et al., 2002, Cordero et al., 2007, Jacinto et al., 2000, Oda and Tsukita, 1999, Ribeiro et al., 2004, Bradley et al., 2003, Jung et al., 2006, Vidal et al., 2006, Chanut-Delalande et al., 2006, Johnson et al., 2011, Caussinus et al., 2008, Shindo et al., 2008, Cabernard and Affolter, 2005, Haghayeghi et al., 2010, Johnson et al., 2008, Gettings et al., 2010, Sekyrova et al., 2010, Kaido et al., 2009, Dottermusch-Heidel et al., 2012, Förster and Luschnig, 2012, Seppa et al., 2008)
|Phenotype Manifest In|
Expression of Scer\GAL4[GMR.PF] driven α-Cat[Scer\UAS.T:Avic\GFP-sg] does not effect patterning of the eye compared to Scer\GAL4[GMR.PF] alone.
Expression of α-Cat[Scer\UAS.T:Avic\GFP-sg] under the control of Scer\GAL4[GMR.PF] results in low level failure to switch cone-cell contacts.
No morphological abnormalities are seen in embryos expressing α-CatScer\UAS.T:Avic\GFP-sg under the control of Scer\GAL4en-e16E.
α-CatScer\UAS.T:Avic\GFP-sg, Scer\GAL4GMR.PF, Scer\GAL4GMR.PF is an enhancer of neuroanatomy defective phenotype of p120ctn308
|Phenotype Manifest In|
α-CatScer\UAS.T:Avic\GFP-sg, Df(2R)E2/shg2, Scer\GAL4btl.PS has embryonic/larval dorsal trunk phenotype
Expression of Scer\GAL4[GMR.PF] driven α-Cat[Scer\UAS.T:Avic\GFP-sg] leads to a mild suppression of the N[fa-g] pupal retina phenotypes. Expression of Scer\GAL4[GMR.PF] driven α-Cat[Scer\UAS.T:Avic\GFP-sg] leads to a mild enhancement of some of the rst pupal retina patterning phenotypes. Expression of Scer\GAL4[GMR.PF] driven α-Cat[Scer\UAS.T:Avic\GFP-sg] leads to a mild enhancement of the p120ctn pupal retina patterning error phenotypes.
Expression of α-Cat[Scer\UAS.T:Avic\GFP-sg] strongly enhances the cone cell switching phenotype seen 41-42 hours after pupal formation when pyd[3.dsRNA.Scer\UAS] is expressed under the control of Scer\GAL4[GMR.PF]. A greater proportion of ommatidia maintain the immature A/P configuration rather than switching to the E/P configuration as is seen in controls.
shg2/Df(2R)E2 embryos with α-CatScer\UAS.T:Avic\GFP-sg driven by Scer\GAL4btl.PS, show largely disordered tracheal branching. No successful dorsal trunk fusions are seen in these embryos. The addition of shgScer\UAS.cOa to these flies rescues these phenotypes to nearly wild-type. Tracheal patterning is normal, although tracheal branches and connectives in the embryos are less extended than in wild-type.
|Complementation & Rescue Data|
|Stocks ( 1 )|
|Notes on Origin|
|External Crossreferences & Linkouts|
|Synonyms & Secondary IDs ( 2 )|
|Secondary FlyBase IDs|
|References ( 33 )|
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|Recent research papers ( 3 )|