Approximately 21% of muscle 12 in stan^E59/stan⁷² larvae have ectopic type 1 synapses, compared to 1.5% in controls. Approximately 14.8% of stan^E59/stan⁷² larval muscle 13 have ectopic type 1 synapses.

6.75% of segmental nerves from stan^E59/stan⁷², show axonal varicosities.

8.6% of muscle 12, 4.7% of muscle 13, and 2% of muscles 6/7 of 3rd instar stan^E59/stan⁷² do not have any synaptic innervation.

Single homozygous dorsal cluster multiple dendritic (MD) neurons (induced in embryos using the MARCM system in a heterozygous background) overextend one or more dendritic processes towards the dorsal midline in about 9% of cases. The basic architecture of the dendritic branching pattern is not altered. Multiple dendritic (MD) neuron dorsal dendritic processes have already extended in 34% of hemisegments in homozygous embryos 11-12 hours after egg laying (in contrast to wild-type embryos where the dorsal dendrites of the MD neurons have not yet extended at this stage).

Mutant embryos exhibit a dorsal dendrite overextension phenotype. 17 hours after egg laying (AEL) the overextension of some dendrites of the multidendritic (md) neuron are already apparent, before the extension of lateral dendrites. Then overextension appears to be only in the dorsal direction. In contrast to wild-type embryos, the shape of the tip structure changes rapidly while the dendritic branch elongates roughly at 0.3μm per minute. Laser ablation experiments demonstrate that, unlike wild-type md neurons, homologous md neurons in contralateral clusters have overlapping fields, suggesting that competition between these dendrites is abolished.

Exhibits excessive dorsal branch outgrowth and misrouting in the dorsal cluster of embryonic dendrites. In 70% of embryos, the dorsal branches over-extend beyond the dorsal midline, intermingling with dorsal branches from the corresponding cluster in the other hemisegment. In addition dorsal dendrites exhibit mis-routing defects and extend branches into the normally vacant space between the anterior and dorsal branches. A similar phenotype is still seen in stan^E59/stan⁷² and stan^E59/stan⁷² embryos. There is no gross defect in muscle patterning or in mid-line epidermal cells.