Co-expression of shgScer\UAS.T:Avic\GFP-rs under the control of Scer\GAL4nos.UTR.T:Hsim\VP16 does not suppress the decreased germline stem cell numbers present in the testis of adults expressing either MtorGD13871 or MtorHMS00735 under the control of Scer\GAL4nos.UTR.T:Hsim\VP16.
Co-expression of shgScer\UAS.T:Avic\GFP-rs with PakScer\UAS.T:Myr1 under the control of Scer\GAL4AB1 completely suppresses the bulging phenotype, and partially suppresses the PakScer\UAS.T:Myr1 salivary gland lumen expansion phenotype.
Co-expression of shgScer\UAS.T:Avic\GFP-rs does not significantly change the impaired epidermal cell elongation phenotype seen during dorsal closure stages in embryos expressing Rab11S25N.Scer\UAS under the control of Scer\GAL4en-e16E.
Ectopic expression of shgScer\UAS.T:Avic\GFP-rs strongly suppresses the Scer\GAL4GMR.PF>cindrdsRNA.PC.PD.Scer\UAS mispatterning phenotype and reduces the distance between interommatidial precursor cells.
Expression of shgScer\UAS.T:Avic\GFP-rs strongly enhances the cone cell switching phenotype seen 41-42 hours after pupal formation when pyd3.dsRNA.Scer\UAS is expressed under the control of Scer\GAL4GMR.PF. A greater proportion of ommatidia maintain the immature A/P configuration rather than switching to the E/P configuration as is seen in controls.
Expression of shgScer\UAS.T:Avic\GFP-rs partially suppresses the tracheal defects seen when Src42ACA.Scer\UAS is expressed under the control of Scer\GAL4btl.PS. Tracheal cells maintain apico-basal polarity for at least two hours longer than when Src42ACA.Scer\UAS is expressed alone, and dorsal branch extension is also partially restored.
The defects in the left-right asymmetry of the hindgut which are seen in homozygous shgR758 embryos are rescued by expression of shgScer\UAS.T:Avic\GFP-rs under the control of either Scer\GAL4mat.αTub67C.T:Hsim\VP16 or Scer\GAL4455.2, but are not rescued by expression of shgScer\UAS.T:Avic\GFP-rs under the control of either Scer\GAL448Y or Scer\GAL4how-24B.