Allele Dmel\N^{intra.GS.Scer\UAS}

General Information
Symbol	Dmel\N^{intra.GS.Scer\UAS}	Species	D. melanogaster
Name		FlyBase ID	FBal0104072
Feature type	allele	Associated gene	Dmel\N

Allele class
Mutagen	in vitro construct - regulatory fusion
Recent Updates
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FB2013_03
FB2013_02
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Nature of the Allele
Allele class
Mutagen	in vitro construct - regulatory fusion (Zaffran and Frasch, 2000)
Mutations Mapped to the Genome

Type Location Additional Notes References
Associated Sequence Data
DDBJ / EMBL / GenBank	DNA sequence Protein sequence Name

UniProtKB/Swiss-Prot
UniProtKB/TrEMBL

Progenitor genotype
Nature of the lesion	Statement Reference Construct: Scer\UAS regulatory sequences drive expression of the intracellular domain of N. (Zaffran and Frasch, 2000)
Carried in construct	P{UAS-N.intra.GS} (Chung and Struhl, 2001, del Alamo et al., 2002, Fusse and Hoch, 2002, Sato and Tomlinson, 2007, Giraldez et al., 2002, Bai and Montell, 2002, Quan et al., 2004, Fuss et al., 2004, Giraldez and Cohen, 2003, Kerber et al., 2001, Zaffran and Frasch, 2000, Hassan et al., 2000, Campbell, 2005, O'Connor-Giles and Skeath, 2003, Song et al., 2007, Vied and Kalderon, 2009, Weng et al., 2010, Vaccari et al., 2008, Araújo and Casanova, 2011, Vaccari et al., 2010, Haenfler et al., 2012, Xiao et al., 2012, Weng et al., 2012)
Cytology
Phenotypic Data
Phenotypic Class
	hyperplasia, with Scer\GAL4^dpp.blk1 (Giraldez and Cohen, 2003) hyperplasia \| cell non-autonomous \| somatic clone, with Scer\GAL4^αTub84B.PL (Giraldez and Cohen, 2003) hyperplasia \| somatic clone, with Scer\GAL4^αTub84B.PL (Giraldez and Cohen, 2003) increased cell number \| larval stage \| somatic clone, with Scer\GAL4^Act5C.PP (Vaccari et al., 2010) neuroanatomy defective, with Scer\GAL4^arm.PS (Giraldez et al., 2002) neuroanatomy defective, with Scer\GAL4^GMR9D11 (Weng et al., 2010) neuroanatomy defective \| larval stage, with Scer\GAL4^wor.PA (Xiao et al., 2012) neuroanatomy defective \| larval stage \| somatic clone, with Scer\GAL4^Act5C.PP (Xiao et al., 2012)
Phenotype Manifest In
	bract, with Scer\GAL4^hs.PB (del Alamo et al., 2002) dMP2 neuron, with Scer\GAL4^pros.PMG (O'Connor-Giles and Skeath, 2003) EL neuron, with Scer\GAL4^pros.PMG (O'Connor-Giles and Skeath, 2003) embryonic/larval neuroblast \| ectopic, with Scer\GAL4^GMR9D11 (Weng et al., 2010) embryonic neuroblast, with Scer\GAL4^{F442A.αTub84B.T:Hsim\VP16} (Struhl and Greenwald, 2001) embryonic proventriculus, with Scer\GAL4^fkh.14-3 (Fuss et al., 2004) eye disc \| somatic clone, with Scer\GAL4^Act5C.PP (Vaccari et al., 2010) female germline stem cell \| ectopic, with Scer\GAL4^C587 (Song et al., 2007) female germline stem cell \| supernumerary, with Scer\GAL4^C587 (Song et al., 2007) follicle stem cell \| somatic clone, with Scer\GAL4^Act.PU, Scer\GAL4^e22c, Scer\GAL4^tub.PU (Vied and Kalderon, 2009) follicle stem cell \| somatic clone, with Scer\GAL4^tub.PU (Vied and Kalderon, 2009) germarium cap cell \| ectopic, with Scer\GAL4^C587 (Song et al., 2007) germarium cap cell \| supernumerary, with Scer\GAL4^C587 (Song et al., 2007) lobula, with Scer\GAL4^ato.3.6 (Hassan et al., 2000) macrochaeta, with Scer\GAL4^hs.PB (del Alamo et al., 2002) medulla, with Scer\GAL4^ato.3.6 (Hassan et al., 2000) polar follicle cell \| ectopic, with Scer\GAL4^Act5C.PI (Bai and Montell, 2002) presumptive embryonic/larval central nervous system, with Scer\GAL4^arm.PS (Giraldez et al., 2002) presumptive embryonic/larval peripheral nervous system, with Scer\GAL4^arm.PS (Giraldez et al., 2002) proventriculus inner layer primordium, with Scer\GAL4^fkh.14-3 (Fuss et al., 2004) proventriculus intermediate layer primordium, with Scer\GAL4^fkh.14-3 (Fuss et al., 2004) RP2 motor neuron, with Scer\GAL4^pros.PMG (O'Connor-Giles and Skeath, 2003) RP2sib neuron \| supernumerary, with Scer\GAL4^pros.PMG (O'Connor-Giles and Skeath, 2003) type II neuroblast \| larval stage \| supernumerary, with Scer\GAL4^wor.PA (Xiao et al., 2012) type I neuroblast \| larval stage \| somatic clone \| supernumerary, with Scer\GAL4^Act5C.PP (Xiao et al., 2012) U neuron \| supernumerary, with Scer\GAL4^pros.PMG (O'Connor-Giles and Skeath, 2003) Usib neuron, with Scer\GAL4^pros.PMG (O'Connor-Giles and Skeath, 2003) vMP2 neuron \| supernumerary, with Scer\GAL4^pros.PMG (O'Connor-Giles and Skeath, 2003) wing disc \| P-stage, with Scer\GAL4^dpp.blk1 (Giraldez and Cohen, 2003)
Detailed Description
	Statement Reference Type II neuroblast clones in the larval brain expressing N[intra.GS.Scer\UAS] under the control of Scer\GAL4[wor.PA] contain mostly supernumerary neuroblasts and a few immature intermediate neural progenitor cells when analysed 72 hours after clone induction. Type I neuroblast clones in the larval brain expressing N[intra.GS.Scer\UAS] under the control of Scer\GAL4[Act5C.PP] contain an average of six neuroblasts per clone, although only 60% of the clones contain more than one neuroblast per clone. (Xiao et al., 2012) Tracheal expression of N[intra.GS.Scer\UAS] under the control of Scer\GAL4[btl.PS] prevents tracheal fusion, but does not arrest outgrowth or stall cells near the base of the ganglionic branch. (Araújo and Casanova, 2011) Expression of N[intra.GS.Scer\UAS] in the eye disc under the control of Scer\GAL4[Act5C.PP] results in clones that overproliferate compared to wild type cells. (Vaccari et al., 2010) Expression of N[intra.GS.Scer\UAS] using Scer\GAL4[CG31670.R9D11] transforms Intermediate Neural Progenitors into ectopic type II neuroblasts in larval brains. (Weng et al., 2010) Follicle stem cell clones expressing N[intra.GS.Scer\UAS] under the control of Scer\GAL4[tub], Scer\GAL4[Act] and Scer\GAL4[e22c] simultaneously are lost more rapidly from the ovarioles than control clones over time. Follicle stem cell clones expressing N[intra.GS.Scer\UAS] under the control of Scer\GAL4[tub] are lost more rapidly from the ovarioles than control clones over time. (Vied and Kalderon, 2009) Expression of N^{intra.GS.Scer\UAS} under the control of Scer\GAL4^C587 results in an increased number of cap cells in the female germarium. Extra cap cells are seen both at the normal location close to the terminal filament cells and also in ectopic locations away from the germarial tip. In germaria with increased numbers of cap cells in the normal location, there is also an increase in the number of spectrosome-containing single germ cells, which behave like germline stem cells (GSCs), at the germarial tip. The ectopically located cap cells appear to be functional as spectrosome-containing single germ cells, which behave as GSCs, are also closely associated with them. These ectopic GSCs anchor their spectrosome on the side that anchors the ectopic cap cell, as occurs in a normal GSC context. Some of the ectopic cap cells are surrounded by inner germarial sheath (IGS) cells or somatic follicle cells. Germ cells lying one cell away from the ectopic GSCs are often germline cysts (as they contain branched fusomes), indicating that the progeny of the ectopic GSCs can probably undergo normal differentiation. However, too many cap cells (more than seven) at either the normal or ectopic location often cause the accumulation of spectrosome-containing single germ cells (which resemble GSCs) located two or more cell diameters away. When N^{intra.GS.Scer\UAS} is expressed under the control of Scer\GAL4^Act5C.PI in inner germarial sheath cells of the adult ovary does not affect the differentiation status of the underlying germ cells. (Song et al., 2007) N^{intra.GS.Scer\UAS}; Scer\GAL4^fkh.14-3 causes ectopic cell movements in the developing proventriculus, but no obvious changes in endodermal or ectodermal cell fate. (Fuss et al., 2004) N^{intra.GS.Scer\UAS}; Scer\GAL4^dpp.blk1 results in overgrowth of the wing pouch and notum and an increase in BrdU incorporation in both of these regions. Similar results are seen in N^{intra.GS.Scer\UAS}; Scer\GAL4^αTub84B.PL somatic clones. These clones also induce overgrowth non-autonomously. Flow cytometry analysis of cell cycle progression in the mutant cells shows no difference in profile compared to wild-type cells. (Giraldez and Cohen, 2003) N^{intra.GS.Scer\UAS}; Scer\GAL4^pros.PMG 30% of RP2 neurons are transformed into RP2 sib neurons, 99% of dMP2 neurons are converted to vMP2, 92% of hemi-segments exhibit an increase in U neurons and there is am overall decrease in EL neurons. (O'Connor-Giles and Skeath, 2003) Expression of N^{intra.GS.Scer\UAS} under the control of Scer\GAL4^Act5C.PI results in the formation of ectopic polar cells near the two poles of the egg chambers. (Bai and Montell, 2002) When N^{intra.GS.Scer\UAS} is driven by Scer\GAL4^hs.PB and heatshocked between 8-12 hours APF no effect is seen on bristle pattern, but a high number of bristles lack bracts. Heat shock at earlier stages strongly reduces the number of bristles. Heatshock at later stages, causes many bristles to lack both shaft and bract. (del Alamo et al., 2002) Embryos expressing N^{intra.GS.Scer\UAS} under the control of Scer\GAL4^arm.PS show loss of peripheral nervous system neurons and the central nervous system is reduced to irregular patches of tissue. (Giraldez et al., 2002) N^{intra.GS.Scer\UAS} when driven by Scer\GAL4^{F442A.αTub84B.T:Hsim\VP16} suppresses neuroblast segregation in the embryo. (Struhl and Greenwald, 2001) Expression of N^{intra.GS.Scer\UAS} under the control of Scer\GAL4^ato.3.6 results in a severe inhibition of axonal branching over the lobula and a complete failure of innervation of the medulla. (Hassan et al., 2000)
External Data
Linkouts
Interactions
	Show the genetic interaction network for Enhancers & Suppressors
Phenotypic Class
Enhanced by
Statement Reference N^{intra.GS.Scer\UAS}, Scer\GAL4^Act5C.PP has neuroanatomy defective \| somatic clone \| larval stage phenotype, enhanceable \| somatic clone by klu^{Scer\UAS.T:Ivir\HA1}, Scer\GAL4^Act5C.PP (Xiao et al., 2012)
Suppressed by
Statement Reference N^{intra.GS.Scer\UAS}, Scer\GAL4^Act5C.PP has neuroanatomy defective \| somatic clone \| larval stage phenotype, suppressible by klu^unspecified/klu^unspecified (Xiao et al., 2012) N^{intra.GS.Scer\UAS}, Scer\GAL4^GMR9D11/Scer\GAL4^erm.R9D11 has neuroanatomy defective phenotype, suppressible by erm^{Scer\UAS.T:Ivir\HA1}, Scer\GAL4^GMR9D11/Scer\GAL4^erm.R9D11 (Weng et al., 2010) N^{intra.GS.Scer\UAS}, Scer\GAL4^wor.PA has neuroanatomy defective \| larval stage phenotype, suppressible by klu^unspecified/klu^unspecified (Xiao et al., 2012)
NOT suppressed by
Statement Reference N^{intra.GS.Scer\UAS}, Scer\GAL4^Act5C.PP has increased cell number \| somatic clone \| larval stage phenotype, non-suppressible by Vha55^j2E9 (Vaccari et al., 2010)
Suppressor of
Statement Reference Scer\GAL4^Act5C.PP/N^{intra.GS.Scer\UAS} is a suppressor of decreased cell number \| somatic clone \| larval stage phenotype of Vha55^j2E9 (Vaccari et al., 2010)
Phenotype Manifest In
Enhanced by
Statement Reference N^{intra.GS.Scer\UAS}, Scer\GAL4^Act5C.PP has type I neuroblast \| supernumerary \| somatic clone \| larval stage phenotype, enhanceable \| somatic clone by klu^{Scer\UAS.T:Ivir\HA1}, Scer\GAL4^Act5C.PP (Xiao et al., 2012)
NOT Enhanced by
Statement Reference N^{intra.GS.Scer\UAS}, Scer\GAL4^ato.3.6 has lobula phenotype, non-enhanceable by ato^Scer\UAS.cJa, Scer\GAL4^ato.3.6 (Hassan et al., 2000) N^{intra.GS.Scer\UAS}, Scer\GAL4^ato.3.6 has medulla phenotype, non-enhanceable by ato^Scer\UAS.cJa, Scer\GAL4^ato.3.6 (Hassan et al., 2000) N^{intra.GS.Scer\UAS}, Scer\GAL4^pros.PMG has dMP2 neuron phenotype, non-enhanceable by spdo^G104/spdo^G104 (O'Connor-Giles and Skeath, 2003) N^{intra.GS.Scer\UAS}, Scer\GAL4^pros.PMG has EL neuron phenotype, non-enhanceable by spdo^G104/spdo^G104 (O'Connor-Giles and Skeath, 2003) N^{intra.GS.Scer\UAS}, Scer\GAL4^pros.PMG has RP2 motor neuron phenotype, non-enhanceable by spdo^G104/spdo^G104 (O'Connor-Giles and Skeath, 2003) N^{intra.GS.Scer\UAS}, Scer\GAL4^pros.PMG has RP2sib neuron \| supernumerary phenotype, non-enhanceable by spdo^G104/spdo^G104 (O'Connor-Giles and Skeath, 2003) N^{intra.GS.Scer\UAS}, Scer\GAL4^pros.PMG has U neuron \| supernumerary phenotype, non-enhanceable by spdo^G104/spdo^G104 (O'Connor-Giles and Skeath, 2003) N^{intra.GS.Scer\UAS}, Scer\GAL4^pros.PMG has Usib neuron phenotype, non-enhanceable by spdo^G104/spdo^G104 (O'Connor-Giles and Skeath, 2003) N^{intra.GS.Scer\UAS}, Scer\GAL4^pros.PMG has vMP2 neuron \| supernumerary phenotype, non-enhanceable by spdo^G104/spdo^G104 (O'Connor-Giles and Skeath, 2003)
Suppressed by
Statement Reference N^{intra.GS.Scer\UAS}, Scer\GAL4^Act5C.PP has type I neuroblast \| supernumerary \| somatic clone \| larval stage phenotype, suppressible by klu^unspecified/klu^unspecified (Xiao et al., 2012) N^{intra.GS.Scer\UAS}, Scer\GAL4^GMR9D11/Scer\GAL4^erm.R9D11 has embryonic/larval neuroblast \| ectopic phenotype, suppressible by erm^{Scer\UAS.T:Ivir\HA1}, Scer\GAL4^GMR9D11/Scer\GAL4^erm.R9D11 (Weng et al., 2010) N^{intra.GS.Scer\UAS}, Scer\GAL4^wor.PA has type II neuroblast \| supernumerary \| larval stage phenotype, suppressible by klu^unspecified/klu^unspecified (Xiao et al., 2012)
NOT suppressed by
Statement Reference N^{intra.GS.Scer\UAS}, Scer\GAL4^Act5C.PP has eye disc \| somatic clone phenotype, non-suppressible by Vha55^j2E9 (Vaccari et al., 2010) N^{intra.GS.Scer\UAS}, Scer\GAL4^ato.3.6 has lobula phenotype, non-suppressible by ato^Scer\UAS.cJa, Scer\GAL4^ato.3.6 (Hassan et al., 2000) N^{intra.GS.Scer\UAS}, Scer\GAL4^ato.3.6 has medulla phenotype, non-suppressible by ato^Scer\UAS.cJa, Scer\GAL4^ato.3.6 (Hassan et al., 2000) N^{intra.GS.Scer\UAS}, Scer\GAL4^pros.PMG has dMP2 neuron phenotype, non-suppressible by spdo^G104/spdo^G104 (O'Connor-Giles and Skeath, 2003) N^{intra.GS.Scer\UAS}, Scer\GAL4^pros.PMG has EL neuron phenotype, non-suppressible by spdo^G104/spdo^G104 (O'Connor-Giles and Skeath, 2003) N^{intra.GS.Scer\UAS}, Scer\GAL4^pros.PMG has RP2 motor neuron phenotype, non-suppressible by spdo^G104/spdo^G104 (O'Connor-Giles and Skeath, 2003) N^{intra.GS.Scer\UAS}, Scer\GAL4^pros.PMG has RP2sib neuron \| supernumerary phenotype, non-suppressible by spdo^G104/spdo^G104 (O'Connor-Giles and Skeath, 2003) N^{intra.GS.Scer\UAS}, Scer\GAL4^pros.PMG has U neuron \| supernumerary phenotype, non-suppressible by spdo^G104/spdo^G104 (O'Connor-Giles and Skeath, 2003) N^{intra.GS.Scer\UAS}, Scer\GAL4^pros.PMG has Usib neuron phenotype, non-suppressible by spdo^G104/spdo^G104 (O'Connor-Giles and Skeath, 2003) N^{intra.GS.Scer\UAS}, Scer\GAL4^pros.PMG has vMP2 neuron \| supernumerary phenotype, non-suppressible by spdo^G104/spdo^G104 (O'Connor-Giles and Skeath, 2003)
Suppressor of
Statement Reference N^{intra.GS.Scer\UAS} is a suppressor of macrochaeta & wing \| ectopic phenotype of Mmus\Neurog1^Scer\UAS.cQa, Scer\GAL4^dpp.blk1 (Quan et al., 2004) Scer\GAL4^Act5C.PP/N^{intra.GS.Scer\UAS} is a suppressor of eye disc \| somatic clone phenotype of Vha55^j2E9 (Vaccari et al., 2010) Scer\GAL4^{F442A.αTub84B.T:Hsim\VP16}/N^{intra.GS.Scer\UAS} is a suppressor of embryonic neuroblast \| supernumerary phenotype of Psn^C2 (Struhl and Greenwald, 2001)
NOT Suppressor of
Statement Reference Scer\GAL4^btl.PS/N^{intra.GS.Scer\UAS} is a non-suppressor of embryonic/larval ganglionic branch phenotype of seq^Z1241 (Araújo and Casanova, 2011) Scer\GAL4^dpp.blk1/N^{intra.GS.Scer\UAS} is a non-suppressor of joint phenotype of TfAP-2^unspecified (Kerber et al., 2001) Scer\GAL4^tub.PU/N^{intra.GS.Scer\UAS} is a non-suppressor \| somatic clone of follicle stem cell \| somatic clone phenotype of mam⁸ (Vied and Kalderon, 2009)
Other
Statement Reference N^{intra.GS.Scer\UAS}, Psn^C2, Scer\GAL4^{F442A.αTub84B.T:Hsim\VP16} has wing \| ectopic phenotype (Struhl and Greenwald, 2001, Struhl and Greenwald, 2001)
Additional Comments
Genetic Interactions
Statement Reference Homozygosity for klu[unspecified] significantly reduces the number of supernumerary neuroblasts and restores intermediate neural progenitor cells and ganglion mother cells in type II neuroblast clones in the larval brain expressing N[intra.GS.Scer\UAS] under the control of Scer\GAL4[wor.PA] (analysed 72 hours after clone induction). Type I neuroblast clones in the larval brain co-expressing klu[Scer\UAS.T:Ivir\HA1] and N[intra.GS.Scer\UAS] under the control of Scer\GAL4[Act5C.PP] contain an average of 18 neuroblasts per clone and 100% of the clones show the supernumerary neuroblast phenotype. Homozygosity for klu[unspecified] significantly reduces the number of supernumerary neuroblasts per clone as well as the frequency of the clones containing more than one neuroblast in type I neuroblast clones in the larval brain expressing N[intra.GS.Scer\UAS] under the control of Scer\GAL4[Act5C.PP]. (Xiao et al., 2012) Expression of N[intra.GS.Scer\UAS] under the control of Scer\GAL4[btl.PS] in seq[Z1241] mutant trachea is not sufficient to suppress the ganglionic branch bifurcation phenotype. Activation of N in tracheal cells does not inhibit migration of ganglionic branch cells and is not able to overcome the effects of higher FGF in the ganglionic branches surrounding tissues of seq[Z1241] mutants. (Araújo and Casanova, 2011) A Vha55[j2E9] mutant clone background does not suppress the overproliferation phenotype seen when N[intra.GS.Scer\UAS] is expressed in eye disc clones under the control of Scer\GAL4[Act5C.PP]. (Vaccari et al., 2010) Coexpression of erm[Scer\UAS.T:Ivir\HA] under the control of Scer\GAL4[CG31670.R9D11] suppresses the ectopic neuroblasts induced by expression of N[intra.GS.Scer\UAS]. (Weng et al., 2010) Expression of N[intra.GS.Scer\UAS] under the control of Scer\GAL4[tub] has no effect on the maintenance of mam[8] follicle stem cell clones. The ability of ptc[S2] to induce follicle stem cell duplications in clones and for the clones to take over the whole ovariole is suppressed by expression of N[intra.GS.Scer\UAS] under the control of Scer\GAL4[tub]. The loss of follicle stem cell clones caused by expression of N[intra.GS.Scer\UAS] under the control of Scer\GAL4[tub] is not substantially suppressed by ptc[S2]. (Vied and Kalderon, 2009) N^{intra.GS.Scer\UAS}; Scer\GAL4^αTub84B.PL; wg^l-17/wg^l-17 somatic clones in the wing disc overproliferate, and non-autonomously induce overproliferation. However, unlike when such clones are wg⁺, this phenotype is mainly restricted to clones in the hinge and pleura, and is rarely seen in clones located in proximal regions of the wing pouch. Flow cytometry analysis of cell cycle progression in the mutant cells shows no difference in profile compared to wild-type cells. (Giraldez and Cohen, 2003) The transformation of neurons (RP2 to RP2sib; dMP2 to vMP2; Usib to U) seen in N^{intra.GS.Scer\UAS}; Scer\GAL4^pros.PMG embryos is unaffected by spdo^G104/spdo^G104. (O'Connor-Giles and Skeath, 2003) In the absence of AP-2 activity, activated N is not able to rescue joint formation. (Kerber et al., 2001) Psn^C2 clones that also express N^{intra.GS.Scer\UAS} lead to double dorsal wing outgrowth. (Struhl and Greenwald, 2001)
Xenogenetic Interactions
Statement Reference
Complementation & Rescue Data
Comments
Stocks ( 0 )

Notes on Origin
Discoverer	G. Struhl

External Crossreferences & Linkouts
Other Crossreferences

Linkouts

Synonyms & Secondary IDs ( 2 )
Reported As
Symbol Synonym	N^{intra.GS.Scer\UAS} N^intra (Quan et al., 2004)
Name Synonym
Secondary FlyBase IDs

References ( 24 )

Generate a list of	All references relating to this allele ( 24 )

List References by type	Research paper ( 24 )
Recent research papers ( 4 )
	Haenfler et al., 2012, Dev. Biol. 365(1): 219--228 Cortical aPKC kinase activity distinguishes neural stem cells from progenitor cells by ensuring asymmetric segregation of Numb. [FBrf0218950] Weng et al., 2012, Dev. Neurobiol. 72(11): 1376--1390 Changes in Notch signaling coordinates maintenance and differentiation of the Drosophila larval optic lobe neuroepithelia. [FBrf0219799] Xiao et al., 2012, Development 139(15): 2670--2680 klumpfuss distinguishes stem cells from progenitor cells during asymmetric neuroblast division. [FBrf0218804] Araújo and Casanova, 2011, J. Cell Sci. 124(14): 2335--2340 Sequoia establishes tip-cell number in Drosophila trachea by regulating FGF levels. [FBrf0214024] Show all research papers ...

Allele Dmel\Nintra.GS.Scer\UAS

Allele Dmel\N^{intra.GS.Scer\UAS}