mushroom body clones have normal axon pruning.
homozygous clones in the dorsal air sac primordium are found at the tip of the primordium at a significantly lower frequency than wild-type clones.
Embryos derived from homozygous female germ-line clones display terminal defects. Most embryos fail to hatch and show a reduction of posterior Filzkorper and the 8th abdominal segment as well as mild defects in the anterior head skeleton. Malpighian tubules are always absent. Embryos lacking both maternal and zygotic Shc
function show defects in germband retraction and a variable reduction of ventral denticle belts not seen in paternally rescued embryos. 70% of homozygotes die during the late pupal stage. Females that survive to adulthood do not lay eggs. Homozygous adult survivors are delayed in development and show defects in the eye, wing and ovary. The eyes are reduced in size and slightly roughened. The severity of the phenotype is graded in an anterior to posterior fashion, with the anterior side being more disorganised than the posterior. The ommatidia in the posterior region of the eye appear essentially normal and contain the full complement of photoreceptors, whereas in the anterior part of the eye, several ommatidia lack one or more outer photoreceptors. These mutant ommatidia do contain small central R7-like photoreceptors. In some ommatidia, the photoreceptors show rotated planar polarity. There is a partially penetrant loss of wing vein material in the distal part of the L4 vein. Homozygous females contain mature oocytes surrounded by a weakly ventralised chorion (as indicated by the fusion of the dorsal appendages).