vvlScer\UAS.cCa; Scer\GAL4lab.PH partially suppresses the brain phenotype of lab14 homozygous embryos: longitudinal axon pathways in the tritocerebrum are restored but the tritocerebral commissure remains absent and axonal projection of the frontal connective remain abnormal.
The addition of vvlScer\UAS.cCa (driven by Scer\GAL4GH146) to acj66 single cell clones in DL1 leads to a complete failure to innervate the glomerulus DL1. Most of their dendrites partially innervated one or two glomeruli anterior to DL1, with some wandering dendrites within the dorsal half of the antennal lobe. This combination of mutants also leads to a novel stereotyped phenotype in DL1 projection neurons. The dorsal branch has additional side branches; the lateral branch stops well short of the lateral edge of the lateral horn and instead curves and branches to innervate the ventral area of the lateral horn nerver innervated by wild-type DL1 axons.