UAS sequences drive expression of wild-type N.
male genitalia, with Scer\GAL4Abd-B-LDN
Expression of NScer\UAS.cSb under the control of Scer\GAL4c768 has no effect on neuroepithelium growth in the developing optic lobe in larvae.
~54% of males expressing NScer\UAS.cSb under the control of Scer\GAL4Abd-B-LDN show incomplete genitalia rotation.
NScer\FRT.Scer\UAS.T:Ivir\HA1 when driven by Scer\GAL4F442A.αTub84B.T:Hsim\VP16 has little or no effect on neuroblast segregation.
Scer\GAL4Act5C.PU/NUAS.cSb is a suppressor | partially of cell polarity defective | recessive | somatic clone phenotype of flwFP41
NUAS.cSb, Scer\GAL4c768 is a suppressor of larval optic anlage phenotype of RPA3GD4799, Scer\GAL4c768
Scer\GAL4Act5C.PU/NUAS.cSb is a suppressor | partially of oocyte | somatic clone phenotype of flwFP41
Scer\GAL4VP16.F442A.αTub84B/NUAS.cSb is a non-suppressor of embryonic neuroblast | supernumerary phenotype of PsnC2
Co-expression of NScer\UAS.cSb significantly rescues the loss of neuroepithelium cells seen in the optic lobe of third instar larvae expressing RPA3GD4799 under the control of Scer\GAL4c768.
Expression of NScer\UAS.cSb under the control of Scer\GAL4Act5C.PU partially suppresses the oocyte polarity defect seen when the posterior follicle cells are mutant for flwFP41.