leads to 100% lethality in embryonic or early larval stages.
The apical membranes of photoreceptor rhabdomeres are severely disrupted in MoePL54
homozygous somatic clones. The rhabdomere microvillar array is irregular in these cells: the rhabdomere terminal web is replaced by abnormal F-actin accumulations.
A proportion of the eggs laid by heterozygous MoePL54
heterozygous females display segmentation defects (8%, n=64). These abnormal embryos lack posterior structures.