Expression of MICAL^GW.Scer\UAS under the control of Scer\GAL4^elav.PLu results in 36.7% of hemisegments having defects in muscle 6/7 innervation (in 43.5% of these cases they show increased muscle innervation, excessive branching or projection into abnormal target fields) and 44.4% having defects in muscle 12/13 innervation. 48.5% of hemisegments show SNa pathway defects (in 37.8% of these cases they show fasciculation with the ISN, premature branching, follow abnormal pathways or terminate on the wrong muscle). In some cases, axons bypass their muscle targets but then appear to defasciculate in inappropriate places and project into adjacent segments. Severely defasciculated and tangled axons are also seen. Axons are seen projecting along the entire length of the muscle 6/7 cleft and dramatic axonal wandering within muscle fields is also seen.

Mical^GW.UAS, Scer\GAL4^elav-C155 is a non-enhancer of short lived phenotype of Hsap\MAPT^UAS.cWa, Scer\GAL4^elav-C155

Mical^GW.UAS, Scer\GAL4^elav-C155 is a non-suppressor of short lived phenotype of Hsap\MAPT^UAS.cWa, Scer\GAL4^elav-C155