Ectopic expression of RagA-B^{Q61L.Scer\UAS} under the control of the Scer\GAL4^elav-C155 driver does not alter the number of neuromuscular junction boutons in third instar larvae compared to controls.

(Wong et al., 2015)

Expression of RagA^{Q61L.Scer\UAS} using Scer\GAL4^nub-AC-62 results in abnormally large wings in which cell number is increased.

(Resnik-Docampo and de Celis, 2011)

Expression of RagA^{Q61L.Scer\UAS} under the control of Scer\GAL4^en.PU results in an increase in the area of the posterior compartment of the wing (the domain of driver expression) compared to the control anterior compartment.

(Bryk et al., 2010)

Expression of RagA^{Q61L.Scer\UAS} under the control of Scer\GAL4^en-e16E results in a significant increase in the size of the posterior compartment of the wing, compared with that of the control anterior compartment of the wing. The average size of individual cells in the posterior compartment of the wing is significantly increased in these flies. Under starvation conditions, expression of RagA^{Q61L.Scer\UAS} under the control of Scer\GAL4^unspecified in clones in the larval fat body results in a 3-fold increase in cell size compared with neighbouring control cells. Under normal fed conditions, expression of RagA^{Q61L.Scer\UAS} under the control of Scer\GAL4^unspecified in clones in the larval fat body has little effect on cell size. The number of autophagosomes per cell is reduced in clones of fat body cells expressing RagA^{Q61L.Scer\UAS} under the control of Scer\GAL4^unspecified under starvation conditions compared to control cells under these conditions. Expression of RagA^{Q61L.Scer\UAS} under the control of Scer\GAL4^Cg.PA has no significant effect on viability under normal fed conditions, but results in a significantly accelerated death rate under starvation conditions compared to control flies.

(Kim et al., 2008)

RagA-B^Q61L.UAS, Scer\GAL4^nub-AC-62 has size defective phenotype, suppressible by hppy^UAS.cMa, Scer\GAL4^nub-AC-62

(Resnik-Docampo and de Celis, 2011)

RagA-B^Q61L.UAS, Scer\GAL4^nub-AC-62 has increased cell number phenotype, suppressible by hppy^UAS.cMa, Scer\GAL4^nub-AC-62

(Resnik-Docampo and de Celis, 2011)

Scer\GAL4^elav-C155/RagA-B^Q61L.UAS is a suppressor of neuroanatomy defective | third instar larval stage phenotype of Trpml¹

(Wong et al., 2015)

Scer\GAL4^nSyb.PS/RagA-B^Q61L.UAS is a suppressor of neuroanatomy defective | third instar larval stage phenotype of Trpml¹

(Wong et al., 2015)

Scer\GAL4^elav-C155/RagA-B^Q61L.UAS is a suppressor of neuroanatomy defective | third instar larval stage phenotype of Cln3^ΔMB1

(Wong et al., 2015)

Scer\GAL4^elav-C155/RagA-B^Q61L.UAS is a suppressor of partially lethal - majority die phenotype of Trpml¹

(Wong et al., 2015)

Scer\GAL4^elav-C155/RagA-B^Q61L.UAS is a suppressor of some die during pharate adult stage phenotype of Trpml¹

(Wong et al., 2015)

Scer\GAL4^elav-C155/RagA-B^Q61L.UAS is a suppressor of neuromuscular junction | third instar larval stage phenotype of Trpml¹

(Wong et al., 2015)

Scer\GAL4^elav-C155/RagA-B^Q61L.UAS is a suppressor of NMJ bouton | third instar larval stage phenotype of Trpml¹

(Wong et al., 2015)

Scer\GAL4^nSyb.PS/RagA-B^Q61L.UAS is a suppressor of neuromuscular junction | third instar larval stage phenotype of Trpml¹

(Wong et al., 2015)

Scer\GAL4^nSyb.PS/RagA-B^Q61L.UAS is a suppressor of NMJ bouton | third instar larval stage phenotype of Trpml¹

(Wong et al., 2015)

Scer\GAL4^elav-C155/RagA-B^Q61L.UAS is a suppressor of embryonic/larval neuromuscular junction | third instar larval stage phenotype of Cln3^ΔMB1

(Wong et al., 2015)

Scer\GAL4^elav-C155/RagA-B^Q61L.UAS is a suppressor of NMJ bouton | third instar larval stage phenotype of Cln3^ΔMB1

(Wong et al., 2015)

Scer\GAL4^Cg.PA/RagA-B^Q61L.UAS is a suppressor of embryonic/larval fat body | second instar larval stage phenotype of Trpml¹

(Wong et al., 2012)

Scer\GAL4^Cg.PA/RagA-B^Q61L.UAS is a suppressor of embryonic/larval fat body | third instar larval stage phenotype of Trpml¹

(Wong et al., 2012)