Abd-B: Abdominal-B (I. Duncan and S. Celniker)
Heterozygotes for null alleles show weak
anteriorly-directed transformations of A5, A6, and A7. In the
male, this results in the presence of a tiny extra tergite in
A7 and a loss of pigmentation on the A5 tergite. Heterozygotes are partially to completely sterile in both sexes, but
are fertile if a duplication for the BXC [such as Dp(3;5)P5 or
Dp(3;1)P68] is present. Hemizygotes and homozygotes are
lethal; embryos lack posterior spiracles and filzkorper, have
the ventral setal bands of A6, A7 and A8 transformed toward A5
or A4, and develop rudimentary chitinized plates in posterior
iab5: infra-abdominal 5 (I. Duncan)
Hemizygotes show strong transformation of A5 toward
A4, resulting in a loss of black pigment in the A5 tergite of
the male. In addition, A6 may be weakly transformed toward
A4. When homozygous, iab5301 causes a weak transformation of
A3 toward A4 as well as a transformation of A5 to A4.
iab6: infra-abdominal 6 (I. Duncan)
Hemizygotes show strong transformations of both A5
and A6 toward A4. Males show a loss of pigment on the A5 and
A6 tergites and show the development of bristles on the A6
sternite. Some alleles cause weak transformations of A4
iab7: infra-abdominal 7 (I. Duncan)
Hemizygotes show strong transformations of A5, A6,
and A7 toward A4. In males, the A5 and A6 tergites show a
loss of pigmentation and an unpigmented A4-type tergite
develops in A7. Both A6 and A7 show the development of sternites with bristles. Heterozygotes show a small A7 tergite in
the male. Two gain-of-function alleles recorded. iab7Spth
(split thorax) heterozygotes display a longitudinal furrow in
the mesothorax; iab7SGA heterozygotes causes abdominal structures to develop in the back of the head (Awad, Gausz, Gyurkovics, and Parducz, 1981, Acta Biol. Acad. Sci. Hung.
32: 219-28; Kuhn and Packert, 1988, Dev. Biol. 125: 8-18).
iab8: infra-abdominal 8 (I. Duncan and S. Celniker)
Hemizygous adult males show strong transformation of
A5, A6, A7 toward A4. In addition, an A8 tergite develops
which is half the size of a normal tergite. In these males,
A5 and A6 tergites show a loss of pigmentation and an unpigmented A4-type tergite develops in A7. A6, A7, and A8 all
development sternites, the first two with bristles.
iab9: infra-abdominal 9 (I. Duncan and S. Celniker)
Adult homozygotes or hemizygotes show absent or
defective genitalia and analia in both sexes. Adults homozygous or heterozygous for iab965 show in addition a partial
transformation of A6 toward A7. Embryos hemizygous for iab9
mutations show the development of a zone of naked cuticle and
a rudimentary ninth abdominal setal belt posterior to the
eighth abdominal setal belt. Posterior spiracles are absent
in iab965 and iab948 hemizygotes and are defective in
iab9Uab1, iab9tuh-3, and iab9Tab hemizygotes. iab965 and
iab948, but not the other iab9 mutations, cause transformations of the A8 setal belt (located in anterior A8) toward A7.
Mcp: Miscadastral pigmentation (E.B. Lewis)
Mcp homozygotes have the fourth (A4) and fifth (A5)
abdominal segments transformed to a state intermediate between
A5 and A6. Similar, but weaker, tranformations occur in Mcp/+
heterozygotes. Mcp1 can be scored in males by dark pigmentation of the A4 tergite and in females by an effect on the
orientation of the lateral bristles of the A4 tergite.
Sab: Superabdominal (E.B. Lewis)
Sab/+ adults show patchy transformations of A3 and
A4 to A5. Homozygote viable and more extreme than heterozygote. Mcp1 Sab1 homozygotes show transformations of A3, A4,
and A5 to a state intermediate between A5 and A6.
Tab: Transabdominal (E.B. Lewis)
Heterozygous adults have a pair of laterally
disposed longitudinal stripes of tissue on the second thoracic
segment (T2) probably corresponding to tissue from the dorsal
sixth (A6) and/or seventh (A7) abdominal segments. These
ectopic stripes of tissue are entirely black in males but only
partially pigmented in females. Tab/+ flies are virtually
sterile, and have a thin seventh tergite in males. Fertility
is partially restored in the presence of a duplication for
BXC, such as Dp(3;1)P68. When hemizygous, Tab embryos have a
reduction of the posterior spiracles and filzkorper, and a
tiny ninth abdominal ventral setal belt that appears as a
small row of denticles posterior to the A8 setal belt.
Eyes of heterozygote small or absent. Scutellars
curve upward. Viability and fertility good. Homozygote usually more extreme than heterozygote but not reliably distinguishable. ale Mc homozygotes completely eyeless; fertile
except when crossed to each other (Golubovsky and Zakarov,
1972, DIS 49: 112). Viability of homozygote varies from 100
down to 40%. RK1A.