μ3, AP-3, AP3μ3, apm3
Gene model reviewed during 5.45
Gene model reviewed during 5.52
Click to get a list of regulatory features (enhancers, TFBS, etc.) and gene disruptions (point mutations, indels, etc.) within or overlapping Dmel\cm using the Feature Mapper tool.
GBrowse - Visual display of RNA-Seq signalsView Dmel\cm in GBrowse 2
Please Note FlyBase no longer curates genomic clone accessions so this list may not be complete
Please Note This section lists cDNAs and ESTs that fall within the genomic extent of the gene model, which may include cDNAs and ESTs of genes within introns, or of overlapping genes. Please see GBrowse for alignment of the cDNAs and ESTs to the gene model.
For each fully sequenced cDNA the DGRC maintains various forms of the cDNA (e.g tagged or untagged) in several different host vectors for subsequent cloning and expression in Drosophila and Drosophila cell lines.
dsRNA made from templates generated with primers directed against this gene tested in RNAi screen for effects on Kc167 and S2R+ cell morphology.
hobo dysgenesis causes instability at the cm locus, in progeny derived from the unstable chromosome U-7 line. Once cm mutations are produced they are genetically stable and do not revert. Cytological analysis suggests that the mutations involve minute structural aberrations, and molecular analysis revealed that many result from the deletion of DNA between two hobos.
Lesions in cm reduce or eliminate pigmentation in the eyes and ocelli and block pigmentation of the fat body and tubules: cm is required for normal pigmentation of all four tissues.
Mohr, 27th April 1927.