Low-frequency RNA-Seq exon junction(s) not annotated.
Gene model reviewed during 5.45
Gene model reviewed during 5.48
gene_with_stop_codon_read_through ; SO:0000697
Stop-codon suppression (UGA) postulated; FBrf0243886.
Double stop-codon suppression (UGA, UAA) postulated; FBrf0243886.
Gene model reviewed during 6.32
7.3, 5.8, 5.3 (northern blot)
None of the polypeptides share 100% sequence identity.
1990 (aa); 213 (kD predicted)
Click to get a list of regulatory features (enhancers, TFBS, etc.) and gene disruptions (point mutations, indels, etc.) within or overlapping Dmel\px using the Feature Mapper tool.
GBrowse - Visual display of RNA-Seq signalsView Dmel\px in GBrowse 2
Please Note FlyBase no longer curates genomic clone accessions so this list may not be complete
Please Note This section lists cDNAs and ESTs that fall within the genomic extent of the gene model, which may include cDNAs and ESTs of genes within introns, or of overlapping genes. Please see GBrowse for alignment of the cDNAs and ESTs to the gene model.
For each fully sequenced cDNA the DGRC maintains various forms of the cDNA (e.g tagged or untagged) in several different host vectors for subsequent cloning and expression in Drosophila and Drosophila cell lines.
Source for identity of: px CG4444
The px gene product may regulate transcription of vein and intervein genes by tethering transcriptional regulators to specific locations in the nucleus.
Mutations in px allow ectopic venation in the intervein regions of the wing.
Mutants display hyperplastic phenotype, showing tissue overgrowth in mitotic recombination clones.
Mutations in px cause extra longitudinal or transverse veins with plexated vein phenotypes. Px, px, net, dsr and emc belong to the same synergistic group and act synergistically against kn, fu and shf.
Px, net, dsr, px and emc loci belong to the plexus phenotypic group within the 'excess-of-vein' mutant class. Extra veins occur parallel to and between normal veins that connect to each other by crossveins, forming plexi. Plexi are distal. Proximal regions shift to the margin leaving a central area devoid of veins. Sensilla and chaetae appear shifted. Combinations of mutations produce superadditive phenotypes.