l(3)72Dd, DMBS, DMYPT, l(3)03802
regulatory subunit of myosin phosphatase flapwing - involved in dorsal closure - involved in cell movement and in the arrest of constriction of contractile rings and ring canals during oogenesis.
Annotated transcripts do not represent all supported alternative splices within 5' UTR.
Annotated transcripts do not represent all possible combinations of alternative exons and/or alternative promoters.
Low-frequency RNA-Seq exon junction(s) not annotated.
Gene model reviewed during 5.46
Gene model includes transcripts encoding non-overlapping portions of the full CDS.
Gene model reviewed during 6.28
None of the polypeptides share 100% sequence identity.
Click to get a list of regulatory features (enhancers, TFBS, etc.) and gene disruptions (point mutations, indels, etc.) within or overlapping Dmel\Mbs using the Feature Mapper tool.
GBrowse - Visual display of RNA-Seq signalsView Dmel\Mbs in GBrowse 2
Please Note FlyBase no longer curates genomic clone accessions so this list may not be complete
Please Note This section lists cDNAs and ESTs that fall within the genomic extent of the gene model, which may include cDNAs and ESTs of genes within introns, or of overlapping genes. Please see GBrowse for alignment of the cDNAs and ESTs to the gene model.
For each fully sequenced cDNA the DGRC maintains various forms of the cDNA (e.g tagged or untagged) in several different host vectors for subsequent cloning and expression in Drosophila and Drosophila cell lines.
Source for identity of: Mbs l(3)72Dd
The 'l(3)72Dd' (Mbs) complementation group comprises 11 EMS-induced mutant alleles.
Photoreceptor neurons require Mbs to reduce myosin activity and thus prevent photoreceptors from dropping out of the eye disc epithelium and moving toward and through the optic stalk. Mbs reduces myosin activity by dephosphorylating sqh.