Atro, Atrophin, l(3)03928, l(3)01323
transcriptional cofactor for Even-skipped - positive regulator of - homolog of mammalian Atrophin
Please see the JBrowse view of Dmel\Gug for information on other features
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AlphaFold produces a per-residue confidence score (pLDDT) between 0 and 100. Some regions with low pLDDT may be unstructured in isolation.
Annotated transcripts do not represent all possible combinations of alternative exons and/or alternative promoters.
Annotated transcripts do not represent all supported alternative splices within 5' UTR.
Low-frequency RNA-Seq exon junction(s) not annotated.
Gene model reviewed during 5.46
Click to get a list of regulatory features (enhancers, TFBS, etc.) and gene disruptions (point mutations, indels, etc.) within or overlapping Dmel\Gug using the Feature Mapper tool.
Comment: maternally deposited
Gug transcript is detected at all stages of embryogenesis.
GBrowse - Visual display of RNA-Seq signals
View Dmel\Gug in GBrowse 2Atrophin
Please Note FlyBase no longer curates genomic clone accessions so this list may not be complete
Please Note This section lists cDNAs and ESTs that fall within the genomic extent of the gene model, which may include cDNAs and ESTs of genes within introns, or of overlapping genes. Please see GBrowse for alignment of the cDNAs and ESTs to the gene model.
For each fully sequenced cDNA the DGRC maintains various forms of the cDNA (e.g tagged or untagged) in several different host vectors for subsequent cloning and expression in Drosophila and Drosophila cell lines.
Source for merge of: l(3)03928 l(3)01323
Source for merge of: Gug CG6964
Source for merge of: Gug Atro
Gug is crucial for correct segmentation during embryogenesis.
Gug is required in diverse developmental processes including early embryo patterning.
Identification: Modifies tsh expression in imaginal disc.