Low-frequency RNA-Seq exon junction(s) not annotated.
Gene model reviewed during 5.52
Click to get a list of regulatory features (enhancers, TFBS, etc.) and gene disruptions (point mutations, indels, etc.) within or overlapping Dmel\ppk29 using the Feature Mapper tool.
GBrowse - Visual display of RNA-Seq signalsView Dmel\ppk29 in GBrowse 2
Please Note This section lists cDNAs and ESTs that fall within the genomic extent of the gene model, which may include cDNAs and ESTs of genes within introns, or of overlapping genes. Please see GBrowse for alignment of the cDNAs and ESTs to the gene model.
For each fully sequenced cDNA the DGRC maintains various forms of the cDNA (e.g tagged or untagged) in several different host vectors for subsequent cloning and expression in Drosophila and Drosophila cell lines.
The ppk29 gene regulates neuronal excitability by a protein-independent mechanism, by affecting the level of activity of the sei potassium channel. This regulation occurs via a protein-independent mechanism: the 3'UTR of the ppk29 gene acts as a natural antisense transcript (the two genes are convergently transcribed and the 3'UTRs overlap by 88bp) which regulates the neuronal mRNA levels of sei via the canonical endogenous siRNA pathway. This regulatory interaction is not symmetric (the sei 3'UTR does not regulate ppk29 levels).
The gene is named "not passionate" after the reduced courtship phenotype shown by mutant males.