Low-frequency RNA-Seq exon junction(s) not annotated.
Gene model reviewed during 5.53
Gene model reviewed during 5.45
short CDS OK
Gene model reviewed during 5.55
None of the polypeptides share 100% sequence identity.
Click to get a list of regulatory features (enhancers, TFBS, etc.) and gene disruptions (point mutations, indels, etc.) within or overlapping Dmel\RpL10Ab using the Feature Mapper tool.
RpL10Ab protein is localized to the cytoplasm in nurse cells and follicle cells. Expression levels are high in early oogenesis, decreases mid-oogenesis, and increases again at oogenesis stage S10. :RpL10Ab expression is not found in nurse cells after oogenesis stage 12, and is decreased in follicle cells after stage 13.
GBrowse - Visual display of RNA-Seq signalsView Dmel\RpL10Ab in GBrowse 2
Please Note This section lists cDNAs and ESTs that fall within the genomic extent of the gene model, which may include cDNAs and ESTs of genes within introns, or of overlapping genes. Please see GBrowse for alignment of the cDNAs and ESTs to the gene model.
For each fully sequenced cDNA the DGRC maintains various forms of the cDNA (e.g tagged or untagged) in several different host vectors for subsequent cloning and expression in Drosophila and Drosophila cell lines.
Source for merge of CG7283 BcDNA:RH06366 was a shared cDNA ( date:030728 ).
Nonsense-mediated mRNA decay (NMD) down-regulates a distinct splice isoform(s) of this gene.
RNAi screen using dsRNA made from templates generated with primers directed against this gene results in aberrantly short, monopolar spindles when assayed in S2 cells. This phenotype can be observed when the screen is performed with or without Cdc27 dsRNA.
Not a Minute gene.
Deletions removing RpL10Ab but no other cytoplasmic ribosomal protein-encoding genes do not show Minute phenotypes.
RNAi generated by PCR using primers directed to this gene causes a cell growth and viability phenotype when assayed in Kc167 and S2R+ cells.