Gene model reviewed during 5.49
Annotated transcripts do not represent all supported alternative splices within 5' UTR.
Low-frequency RNA-Seq exon junction(s) not annotated.
Gene model reviewed during 5.55
Click to get a list of regulatory features (enhancers, TFBS, etc.) and gene disruptions (point mutations, indels, etc.) within or overlapping Dmel\nemy using the Feature Mapper tool.
nemy is expressed in anterior midline glia but not in posterior midline glia in stage 12 embryos.
nemy transcripts are detected in third instar larval brains in the midline cells, ventral nerve cord, and in a bilaterally paired cell in the abdominal ganglion (A8). Strong signal is also observed in a pair of descending neurons and in a subset of protocerebral neurons. It is partially co-expressed with Phm.
Expression pattern inferred from unspecified enhancer trap line.
Expression is observed in the anterior midline glial cells.
GBrowse - Visual display of RNA-Seq signalsView Dmel\nemy in GBrowse 2
Please Note This section lists cDNAs and ESTs that fall within the genomic extent of the gene model, which may include cDNAs and ESTs of genes within introns, or of overlapping genes. Please see GBrowse for alignment of the cDNAs and ESTs to the gene model.
For each fully sequenced cDNA the DGRC maintains various forms of the cDNA (e.g tagged or untagged) in several different host vectors for subsequent cloning and expression in Drosophila and Drosophila cell lines.
The "nemy" mutant was previously associated with the CG8772 annotation in FlyBase, based on information in GenBank accession AF417120. However, data from FBrf0207202 indicates that the nemy mutant phenotype is due to an effect on the neighbouring CG8776 annotation, and not due to an effect on CG8772. Thus, the CG8772 annotation has been split out into a separate gene report in FlyBase (and has been renamed CG42708 in release 5.26 of the genome annotation to avoid confusion) and the nemy mutant has instead been merged with the gene corresponding to the CG8776 annotation in FlyBase.