Click to get a list of regulatory features (enhancers, TFBS, etc.) and gene disruptions (point mutations, indels, etc.) within or overlapping Dmel\btsz using the Feature Mapper tool.
GBrowse - Visual display of RNA-Seq signalsView Dmel\btsz in GBrowse 2
Please Note FlyBase no longer curates genomic clone accessions so this list may not be complete
Please Note This section lists cDNAs and ESTs that fall within the genomic extent of the gene model, which may include cDNAs and ESTs of genes within introns, or of overlapping genes. Please see GBrowse for alignment of the cDNAs and ESTs to the gene model.
For each fully sequenced cDNA the DGRC maintains various forms of the cDNA (e.g tagged or untagged) in several different host vectors for subsequent cloning and expression in Drosophila and Drosophila cell lines.
Source for identity of: btsz CG31306
Source for merge of: btsz CG17304
Source for merge of: l(3)10418 CG18280
Source for merge of: l(3)10418 BcDNA:GH06647
Source for merge of: l(3)10418 CG14858
Source for merge of: btsz Granulophilin
Annotations CG33555 and CG17304 merged as CG44012 in release 5.47 of the genome annotation. Merge supported by RNA-seq junction data.
Source for identity of btsz CG31306 was sequence comparison ( date:030917 ).
Annotations CG14858 and CG18280 merged as CG31306 in release 3 of the genome annotation.
Source for merge of l(3)10418 BcDNA:GH06647 was a shared cDNA ( date:030728 ).
Embryos injected with dsRNA made from templates generated with primers directed against this gene show defects in the organisation of the epithelium at the end of cellularisation. Gastrulation is completely arrested as the epithelium no longer elongates along the anteroposterior axis due to the epithelial cells losing their columnar organisation and becoming mesenchymal.
dsRNA made from templates generated with primers directed against this gene severely affects gastrulation, resulting in the epithelium failing to extend properly. Defects are either strong or medium; that is, they are visible at the beginning of gastrulation or about 15 minutes later, respectively. The defects are penetrant (80%) and dose dependent.