A Database of Drosophila Genes & Genomes

FB2008_07, released August 8, 2008
 

Reference Report

Reference
Citation Homyk, T., Pye, Q. (1989). Some mutations affecting neural or muscular tissues alter the physiological components of the electroretinogram in Drosophila.  J. Neurogenet. 5: 37--48.
FlyBase ID FBrf0050364
Type of publication Research paper
Offprint Available No
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PubMed ID 2703940
PubMed Abstract Mutants displaying generalized behavioral defects and one mutant having an enzyme deficiency were examined for electroretinogram (ERG) defects. Mutations in nine genes were examined that cause ERG defects. Two, parats4 and slrpD, cause reversibly temperature dependent loss of the off-transients in the ERG. stnC and Tyr-2 cause loss of the on and off-transients. The transient defect in Tyr-2 mapped close to a site shown to affect tyrosinase activity in this strain. Mutations bas, rex and sesD delay recovery from the prolonged depolarization afterpotential. The visual defects of mutations elavjl and nbAEE171 are not complemented by lethal mutations, which, presumably, affect other tissues.
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Language of publication English
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Abbreviation J. Neurogenet.
Title Journal of Neurogenetics
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Volume range 1-
Year range 1983-
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Place of publication London
Language of publication English
ISBN/ISSN 0167-7063
CODEN JLNEDK
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