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Reference
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| Citation |
Herndon, L.A., Wolfner, M.F. (1995). A Drosophila seminal fluid protein, Acp26Aa, stimulates egg laying in females for 1 day after mating. Proc. Natl. Acad. Sci. USA 92(22): 10114--10118. |
| FlyBase ID |
FBrf0084020 |
| Type of publication |
Research paper |
| Offprint Available |
Yes |
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External Crossreferences
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| PubMed ID |
7479736 |
| PubMed Abstract |
Mating triggers behavioral and physiological changes in the Drosophila melanogaster female, including an elevation of egg
laying. Seminal fluid molecules from the male accessory gland are responsible for initial behavioral changes, but persistence
of these changes requires stored sperm. Using genetic analysis, we have identified a seminal fluid protein that is responsible
for an initial elevation of egg laying. This molecule, Acp26Aa, has structural features of a prohormone and contains a region
with amino acid similarity to the egg-laying hormone of Aplysia. Acp26Aa is transferred to the female during mating, where
it undergoes processing. Here we report the generation and analysis of mutants, including a null, in Acp26Aa. Females mated
to male flies that lack Acp26Aa lay fewer eggs than do mates of normal males. This effect is apparent only on the first day
after mating. The null mutation has no other detectable physiological or behavioral effects on the male or the mated female.
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| Biosis |
98570607 |
| Zoological record |
13204006654 |
Associated Information
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| Comments |
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| Text of personal communication |
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Also Published As
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Other Reference Information
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| Secondary IDs |
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| Language of publication |
English |
| Additional language(s) of abstract |
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| ISBN |
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| Place of publication |
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Published In
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| Abbreviation |
Proc. Natl. Acad. Sci. USA |
| Title |
Proceedings of the National Academy of Sciences of the United States of America |
| Authors |
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| Volume range |
1- |
| Year range |
1915- |
| Page range |
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| Publisher |
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| Place of publication |
Washington, DC |
| Language of publication |
English |
| ISBN/ISSN |
0027-8424 |
| CODEN |
PNASA6 |
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Data from Reference
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| Aberrations (1)
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| Alleles (3)
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| Genes (3)
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