|Citation||Gruntenko, N.E., Chentsova, N.A., Andreenkova, E.V., Bownes, M., Segal, D., Adonyeva, N.V., Rauschenbach, I.Y. (2003). Stress response in a juvenile hormone-deficient Drosophila melanogaster mutant apterous56f. Insect Mol. Biol. 12(4): 353--363. (Export to RIS)|
|Publication Type||Research paper|
|PubMed Abstract||The apterous56f (ap56f) mutation leads to increases in juvenile hormone (JH) degradation levels and JH-esterase makes a greater contribution to the increase than JH-epoxide hydrolase. Dopamine levels in ap56f females, but not males, are higher than in wild-type. JH treatment of ap56f and wild-type females decreases their dopamine levels. ap56f females, but not males, produce less progeny. Survival under heat stress is dramatically decreased in ap56f females, but not males. ap56f flies show a stress reaction, as judged by changes in tyrosine decarboxylase and JH-hydrolysing activities, dopamine levels and fertility, but its intensity in the mutant females, but not males, differs significantly from wild-type. Thus, the ap56f mutation causes dramatic changes in female, but not male, metabolism and fitness.|
What does this section display?
What does this section not display?
This section does not currently display links that were removed or gene model changes.
|All updates||Click here to see a list of all updates to this record from FB2010_08 and on.|
|Language of Publication||English|
|Additional Languages of Abstract|
|Also Published As|
|Abbreviation||Insect Mol. Biol.|
|Title||Insect Molecular Biology|
|Data from Reference|