Honda alleles. - Honda, B. (2019)
The following information accompanied stocks donated to the Bloomington Stock Center by Barry Honda, Simon Fraser University. Gfat1[Z1914] and Gfat1[Z352] are EMS-induced missense mutations originating in the Zuker screen (FBrf0179036). Gfat1[Z1914] causes an A to T amino acid change at residue 414. Gfat1[Z352] causes an I to M amino acid change at residue 556. Gfat1[Z1914]/Gfat1[Z352] individuals show Splayed phenotypes (see FBgn0003476). Gfat1[I400-8] introduces an E to stop change at amino acid 535. Gfat1[zep-LP13] introduces a Y to stop change at amino acid 334. zep (FBgn0041086) is allelic to Gfat1 (FBgn0027341). This mutation is currently listed as zep[LP13] (FBal0137025). spok[e-204] and spok[Z712] are loss-of-function spok alleles. spok[Z712] originated in the Zuker screen (FBrf0179036). Gfat2[10A-2] and Gfat2[18A-14] are loss-of-function Gfat2 alleles. Oga[1179] and Oga[526] are loss-of-function Oga alleles. Oga[1179] causes a G to R amino acid change at residue 823. Oga[526] causes a G to E amino acid change at residue 960.