bk1 << tll << bk2
Distal breakpoint is at the junction of subtelomeric and telomeric sequences distal to the end of the reference genome assembly.
The 5' end of this breakpoint corresponds to 3R:20308209 and the 3' end corresponds to 3R:20308213 ; the breakpoint contains a 5bp duplication of genome sequence, in addition to the inversion.
Breakpoint(s) molecularly mapped
A positive correlation between the frequency of In(3R)C in a population and the frequency of individuals carrying extra bristles has been demonstrated. The correlation has also been demonstrated at the individual level.
Homozygous viable. Crossing over in 3R reduced to 1% between centromere and ss, to 0.2% between ss and e; no crossovers between e and tip of 3R recovered except for rare doubles within inversion.
Sturtevant, June 1913.
One of 16 polymorphic inversions identified in the Drosophila Genetic Reference Panel (DGRP) freeze 2 lines.
According to Craymer in FBrf0063385, e1 was isolated on In(3R)C and was never successfully separated from it. Most ebony alleles denoted simply as "e" without a specific allele designation can be traced back to Muller's e4. Most ebony mutations in Stock Center stocks designated as e1 are probably e4.
Recovered as: Cosmopolitan inversion.
Isolation: Koleika, Greece.
Naturally occurring inversions have been categorised into classes according to their geographical distribution and frequencies, In(3R)C belongs to the rare Cosmopolitan class.
Distribution in Indian populations studied.
Rare cosmopolitan inversion. The distribution of this inversion in two populations from Valencia, Spain (one from a cellar and one from a vineyard) has been studied.
Rare cosmopolitan inversion (based on Australasian frequencies of distribution).
First inversion demonstrated genetically (Sturtevant, 1926). Used as a balancer for the region from Dl to 3R tip. Balancers contain Sb, e, l(3)a, or l(3)e. Balancer for all of chromosome 3 made by combining with In(3L)P. Found in wild populations (e.g., Oshima and Watanabe, 1965, D. I. S. 40: 88; Ashburner and Lemeunier, 1976, Proc. R. Soc. London, B 193: 137-57; Stalker, 1976, Genetics 82: 323-47; Choi, 1977, D. I. S. 52: 88; 1977, Genetica 47: 155-60).