bk1 hits wb << mol << bk2
From the complementation tests of T(2;4)GT6 against mutations in wb, ms(2)34Fe, l(2)34Fc, l(2)34Fd, Pgant35A, elA, pu and noc it seems likely that T(2;4)GT6 is not a simple two break event, but may carry a duplication of wb-pu in the heterochromatin of the Ts(2Lt;4Lt) component of T(2;4)GT6, in addition to the translocated copies on the Ts(4Lt;2Lt) component. Thus the hypomorphicity of T(2;4)GT6 for wb could be caused by a break that only partially disrupts wb function, or a break that causes a null wb, partially rescued by heterochromatic copies of wb and neighboring genes.
Limits of break 1 from polytene analysis (FBrf0091422) Limits of break 2 from polytene analysis (FBrf0051930)
Hypomorphic for wb, wild type for l(2)34Fc, l(2)34Fd and Pgant35A.
Chromosome acts as a strong trans-suppressor of a physical interaction between tandemly duplicated copies of w+, an interaction that is required for the suppression of genes by z1.