Open Close
General Information
D. melanogaster
FlyBase ID
Feature type
Also Known As
Computed Breakpoints include
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Class of aberration (relative to wild type)
Class of aberration (relative to progenitor)



Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

bk1 << spg << bk2 << l(3)02521

Genetic mapping information
Comments on Cytology

Limits of break 1 from polytene analysis (FBrf0091073) Left limit of break 2 from polytene analysis (FBrf0099763) Right limit of break 2 from polytene analysis (FBrf0091073)

Sequence Crossreferences
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Complementation Data
Partially deleted / disrupted
Molecular Data
Partially deleted
Genes NOT Deleted / Disrupted
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
Molecular Data
Affected Genes Inferred by Location
    Phenotypic Data
    In combination with other aberrations
    NOT in combination with other aberrations

    Df(3R)3450 mutant clones in sensory neurons in adult wing do not display any defects in injury-induced axon degeneration (following an axotomy, the severed axons are cleared away normally).

    Embryos homozygous for Df(3R)3450, which uncovers Cul5, have no detectable defects in tracheal morphology.

    Df(3R)3450/+ adults show a reduction in notal bristles.

    The size of the tracheal lumen is different to wild type in Df(3R)3450 embryos.

    The Df(3R)3450 chromosome does not act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion).

    Heterozygosity for Df(3R)3450 results in 0.0% X chromosome nondisjunction and 0.0% fourth chromosome nondisjunction in In(1)FM7/X ; svspa-pol females.

    No second site non-complementing phenotype with zipEbr and zipmhc-c6.1.

    Shows no maternal enhancement of dpphr4.

    Midgut development of mutant embryos is wild type.

    Stocks (2)
    Notes on Origin

    S. Hayashi.

    Balancer / Genotype Variants of the Aberration
    Separable Components
    Other Comments
    Synonyms and Secondary IDs (9)
    References (83)