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General Information
Symbol
Dp(1;f)26C
Species
D. melanogaster
Name
FlyBase ID
FBab0024566
Feature type
Also Known As
26C
Computed Breakpoints include
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Class of aberration (relative to wild type)
Class of aberration (relative to progenitor)
Breakpoints
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

bk1 << p1:bk1 << p1:bk6 << bk2

Genetic mapping information
Comments
Comments on Cytology

Deletion removing centromere.

Sequence Crossreferences
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Complementation Data
Completely deleted / disrupted
Partially deleted / disrupted
Molecular Data
Completely deleted
Partially deleted
Genes NOT Deleted / Disrupted
Complementation Data
Molecular Data
 
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
 
Molecular Data
 
Affected Genes Inferred by Location
    Phenotypic Data
    In combination with other aberrations

    Dp(1;f)γ878/Dp(1;f)26C heterozygotes increases y+ expression, to 46% of full Dp(1;f)γ158 trans-suppression - relative suppression. y+ expression is quantitated in triple row bristles.

    NOT in combination with other aberrations

    Transmission rate through females to progeny is 1.4% in the first 5 days of egg lay. Transmission is improved in the presence of increased nod+ dosage, provided by P{flag-nodhsp}.

    Transmitted from meiosis at 0.6% in nod3/nod+ females and 2.2% in wild type females.

    Stocks (0)
    Notes on Origin
    Discoverer
     
    Balancer / Genotype Variants of the Aberration
     
    Separable Components
     
    Other Comments
     

    Acentric mini-chromosomes are lost at a moderate but elevated rate during male germline mitotic divisions and in female mitosis, but appear to be transmitted efficiently through pre-blastoderm mitoses and male meiosis. mit(1)15 can localise to the mini-chromosome. Acentric mini-chromosomes are normally inherited, transmission is achieved by a mechanism similar to normal centromere-containing chromosomes, which utilises nod and mit(1)15 and involves movement to the spindle poles. Newly generated acentric fragments from the tip of the normal X chromosome do not display neocentromere activity in male meiosis I.

    Minichromosome is 285kb in length is missing all centric heterochromatin and retaining only euchromatic sequences and subtelomeric heterochromatin.

    Acentric terminal deficiency, 285kb in length containing no centric heterochromatin.

    Monosome transmission behaviour from both male and female parents is highly unstable, all heterochromatin is deleted.

    Synonyms and Secondary IDs (4)
    Reported As
    Name Synonyms
    Secondary FlyBase IDs
    • FBab0024060
    References (9)