The 2R:20274486 release 6 coordinate of the left breakpoint is an estimate. It corresponds to the insertion site of P{EP}CG10444^EP951. Df(2R)BSC19 was generated by expression of P transposase in the presence of trans-heterozygous P elements. Since the deletion chromosome did not retain the miniwhite marker from either progenitor P insertion, it was probably formed by nonhomologous end joining of breaks near the insertion sites of the progenitor P insertions. Df(2R)BSC19 heterozygotes do not show Minute phenotypes, so the haploinsufficient RpS18 gene lies to the left of this breakpoint.

The 2R:20588392 release 6 coordinate of the left breakpoint is an estimate. It corresponds to the insertion site of P{lacW}l(2)s4831^s4831. Df(2R)BSC19 was generated by expression of P transposase in the presence of trans-heterozygous P elements. Since the deletion chromosome did not retain the miniwhite marker from either progenitor P insertion, it was probably formed by nonhomologous end joining of breaks near the insertion sites of the progenitor P insertions.

Fails to complement Df(2R)BSC400.

Inferred to overlap with: Df(2R)BSC400.

Homozygous Df(2R)BSC19 mutant embryos produce a phenotype in which only the dorsal branch of the abdominal segmental nerve (SNa) is absent or malformed. In 25% of abdominal hemisegments one or more of the dorsal branch target muscles is not present, and this could account for the absence of the branch in these hemisegments.

The Df(2R)BSC19 chromosome does not act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{w^var}KR3-2", a stable "brown-red" variant of the P{3'WP-2,w^var}2Lt insertion).