BxJ mutant larvae show reduced haemocyte counts while BxJ/BxJ and BxJ/+ mutants show higher numbers of larval crystal cells when compared to controls.
Homozygotes have small wings lacking much of the wing margin. Heterozygotes show a similar albeit weaker phenotype.
BxA523/BxJ wings are small and lack areas of wing margin. This phenotype is less severe than BxJ wings and is similar to BxJ /+ wings.
Mutant animals exhibit an decreased sensitivity to cocaine. After exposure to 100ug of cocaine, control animals show a slow circling behaviour and some are akinetic, mutant animals are much less affected, showing increased locomotion (mostly in straight lines) decreased slow circling, and almost no akinesia. Mutant animals, unlike controls which show very little movement, remain in motion at 125ug exposure showing circling behaviours.
Homozygotes exhibit tissue loss from the entire wing margin and blistering of the wing blade. The phenotype is unchanged in double homozygote combinations with ct53d.
The only dominant Bx allele not suppressed by a Bx deficiency or hdp.
Clonal analysis of wing disk development indicates massive cell loss during third larval instar. Clones of Bx+ cells in BxJ/+ wings that reach the margin but are confined to the dorsal or ventral surface often cause reconstitution of both surfaces and appearance of marginal elements derived from both surfaces.
Wings reduced to slender strip; only posterior cell present at tip. Heterozygous females have half and homozygous females one third the normal number of cells in membrane of wing. Femur shortened or legs otherwise abnormal, especially third pair. Halteres abnormal. Interacts with bi to give more nearly normal wings. RK1.