Heterozygous eyes are small and rough.
The number of ommatidial clusters is significantly reduced in EgfrE1/+ larval eye discs, resulting in a small rough eye phenotype.
Mutant flies have an extra wing vein phenotype with an additional vein fragment above vein L2 and some vein deltas.
EgfrE1 heterozygous flies display abnormal wing vein morphology. This phenotype is compromised by treatment with the Hsp90 inhibitor 17-AAG.
Heterozygotes have extra wing vein material in the wing.
Heterozygotes have a rough eye phenotype.
In EgfrE1/+ adults, the number of ommatidia with pale-type R8 cells and yellow-type R7 cells is significantly increased. Many of these are found in pale-yellow hybrid ommatidia with a yellow-type R7 and a pale-type R8.
EgfrE1 mutants exhibit ectopic wing veins.
Heterozygotes have a rough eye phenotype, with elliptical eyes and disarranged ommatidial arrays, and wing vein L2 shows an abnormal broadening at the wing margin.
EgfrE1 heterozygotes exhibit a slight rough eye phenotype.
EgfrE1 mutants exhibit ectopic wing vein formation.
EgfrE1 homozygous adults are small and have greatly reduced numbers of ommatidia with large gaps of pigmented tissue between them. Heterozygous adults have normal eyes.
Heterozygous adults have smaller eyes than normal which have moderate rough phenotype.
EgfrE1/+ flies have rough eyes and a weak extra wing vein phenotype.
Wings of heterozygous and homozygous flies have extra wing vein material.
Heterozygotes have smaller eyes than normal which are mispatterned.
EgfrE1/+ flies have a rough eye phenotype.
Heterozygous mutants do not cause any defects in the ocellar sensory system (OSS) neurogenesis or axon guidance.
Heterozygotes have smaller eyes than normal and lack some ommatidia, producing an irregular arrangement of facets.
Heterozygotes have rough eyes. This phenotype is suppressed towards wild-type in transheterozygous combination with Egfrf24. This phenotype is suppressed towards wild-type in transheterozygous combination with Egfrtsla at 28oC but not at 18oC. Heterozygotes show greatly reduced ommatidial cluster formation in the morphogenetic furrow of the eye disc.
Heterozygotes exhibit roughening of patches on both eyes. Homozygotes exhibit strong blistering, including loss of pigmentation, in patches on both eyes. Rhabdomere assembly is disordered with extra material at the apical ends of the ommatidia. Ommatidia pattern is disordered.
Heterozygotes have rough eyes.
Homozygous flies have a few, widely separated ommatidia. Each ommatidium contains a single R8 cell, as in wild-type flies, and this phenotype is not altered in scaWB1/scaOB7 EgfrE3/EgfrE1 double homozygotes.
Increase the penetrance of extra crossveins in chicgdh-5/EgfrE1 heterozygotes.
Heterozygotes have a mild rough eye phenotype due to a disrupted ommatidial array.
Displays locomotor activity rhythm with an approximately 24h period.
Extra wing veins close to the wing margin.
Mutation has no effect on rhohs.sev rough eye phenotype.
Rough eye phenotype: eyes are small and rough at the perimeter, ommatidia and hairs are in an irregular pattern.
Heterozygous rhove-1 dominantly suppresses the homozygous and heterozygous phenotype. Heterozygous rhoWk, rhoMod and rhoStg enhance the heterozygous phenotype.
Homozygous eye discs have fewer ommatidia than wild-type. Outgrowth of photoreceptor axons is disturbed, occurring in many directions. Cell death is abundant in the region approximately corresponding to columns 10-15 in wild-type. In the adult eye the ommatidia are separated by numerous pigment cells and mechanosensory bristles. Wing veins show thickening and plexus formation. Enhances the wing phenotype of Df(1)N-54l9, N264-39, Dl9P, and DlKX6 mutations, and the eye phenotype of Nspl-1.
Mild roughening of the eye.
Homozygotes have fewer ommatidia than wild-type.
The array of ommatidia is less regular in heterozygotes than in wild-type flies, and there is a slight disturbance in the wing vein pattern. Homozygotes have much smaller eyes than normal, containing many fewer ommatidia, and some regions lack ommatidia entirely. These regions contain cells resembling the pigment cells that normally surround each ommatidium, and also mechanosensory bristles. Most ommatidia contain the normal number and arrangement of cells. Only approximately one tenth of the normal number of preclusters differentiate in the eye imaginal discs.
Eyes of EgfrE1/+ heterozygotes rough and more oval than wild type; also display a slight disturbance of the wing-vein pattern. Homozygotes have smaller eyes with many fewer ommatidia and some regions lack them entirely; those ommatidia that are formed contain the normal number and arrangement of cells; the regions without ommatidia contain cells that resemble pigment cells and mechanosensory bristles; only about one tenth the normal number of preommatidial cell clusters differentiate behind the morphogenetic furrow. EgfrE1 in heterozygous combination with a deficiency or null mutation for Egfr is normal in phenotype indicating that EgfrE1 alleles are hypermorphic.