wing margin & macrochaeta | somatic clone
12% of heterozygotes have ectopic or duplicated macrochaetae.
Homozygous clones in the haltere are found infrequently. These clones form vesicles of wing formation segregated inside the haltere. Homozygous clones also cause transformation of the metanotum to mesonotum. Homozygous clones in the posterior wing cause distortions in wing shape; venation is disrupted and large socketed bristles appear along the posterior wing margin. Homozygous clones in the leg cause the femur and tibia to be short and twisted, and the tarsal segments to be enlarged. Leg clones also contain extra bristles. Clones in the anterior first leg are transformed to anterior second leg or have extra sex comb teeth. Clones in the second leg are twisted, have vesicles of extra tissue, or cause the absence of the entire leg. Tarsal segments are often fused, duplicated or absent. Clones in the third leg often have partial or complete duplications of distal structures, or the entire leg morphology is abnormal. Clones in the first and third legs often cause truncations of the leg. Homozygous clones in adult abdominal segments 1 to 6 survive and appear normal. Clones in the 7th abdominal segment appear normal except when associated with abnormalities in the genitalia, including the absence of part of the genital plate or fusions of parts of the genital structures.
mor4 is a suppressor of visible | dominant | adult stage phenotype of Pc4
mor4 has macrochaeta phenotype, enhanceable by brm[+]/brm2
mor[+]/mor4 is an enhancer of macrochaeta phenotype of brm2
mor4 is a suppressor of mesothoracic leg phenotype of Pc4
mor4 is a suppressor of metathoracic leg phenotype of Pc4
brm2, mor[+]/mor4 has ommatidium phenotype
brm2, mor[+]/mor4 has humeral bristle phenotype
brm2, mor4 has ommatidium phenotype
brm2, mor4 has humeral bristle phenotype
Suppresses the extra sex comb phenotype of Pc4, Ts(YLt;2Lt)L124. Causes between 50% and 100% suppression of the Pc4/+ extra sex combs phenotype.