The dorsalised phenotype of eggs derived from kek1RA5/kek1RM2 females is enhanced by one copy of grk+t9. The ventralised phenotype of eggs derived from Egfrt1/Egfrflb-2E07 females is partially suppressed by kek1RA5/kek1RM2.
Extension of longitudinal axons through the intercommissural region is frequently abnormal in kek1RM2; Nrt5 and kek1RA5/kek1RM2 ; Nrt5 embryos. In addition, Fas2 expressing axons show defasciculation, stalling and guidance defects, including crossing of the midline. The stalling and misguidance phenotypes can be rescued by NrtScer\UAS.cSa expressed under the control of Scer\GAL4Mz1277.