Mutants show a short stop phenotype, with defects in the ability of nerve branches to reach target muscles. ISNb (where the effect is most pronounced) and SNa are most sensitive to the loss of trio activity. In addition, there are occasional defects in epidermal muscle attachment sites. The CNS also shows pathfinding errors resulting in breaks in the longitudinal connectives. The axons often fail to traverse the segment boundary.
trio[+]/trio2H is a suppressor of visible phenotype of Rac1GMR.PN
trio[+]/trio2H is a non-suppressor of visible phenotype of Rho1GMR.PH
trio[+]/trio2H is a suppressor of eye phenotype of Rac1GMR.PN
trio[+]/trio2H is a suppressor of ommatidium phenotype of Rac1GMR.PN
trio[+]/trio2H is a non-suppressor of eye phenotype of Rho1GMR.PH
trio[+]/trio2H is a non-suppressor of ommatidium phenotype of Rho1GMR.PH