FB2025_01 , released February 20, 2025
Allele: Dmel\EcRGAL4::LBD.hs
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General Information
Symbol
Dmel\EcRGAL4::LBD.hs
Species
D. melanogaster
Name
FlyBase ID
FBal0137557
Feature type
allele
Associated gene
Associated Insertion(s)
Carried in Construct
Also Known As
hs-GAL4-EcR, hs-GAL4-EcR.LBD
Key Links
Transgenic product class
Nature of the Allele
Transgenic product class
Progenitor genotype
Carried in construct
Cytology
Description

An Hsp70 promoter controls expression of a ligand-inducible transcriptional activator that is composed of the Scer\GAL4 DNA binding domain fused to amino acids 330-878 of EcR (this encodes the ligand binding domain, hinge region and F-domain).

Allele components
Component
Use(s)
Regulatory region(s)
Mutations Mapped to the Genome
Curation Data
Type
Location
Additional Notes
References
Variant Molecular Consequences
Associated Sequence Data
DNA sequence
Protein sequence
 
Expression Data
Reporter Expression
Additional Information
Statement
Reference
 
Marker for
Reflects expression of
Reporter construct used in assay
Human Disease Associations
Disease Ontology (DO) Annotations
Models Based on Experimental Evidence ( 0 )
Disease
Evidence
References
Modifiers Based on Experimental Evidence ( 0 )
Disease
Interaction
References
Comments on Models/Modifiers Based on Experimental Evidence ( 0 )
 
Disease-implicated variant(s)
 
Phenotypic Data
Phenotypic Class
Phenotype Manifest In
Detailed Description
Statement
Reference

Animals expressing EcRhs.T:Scer\GAL4 have defects in the mushroom body α/β lobes: the lobes may appear slim or underdeveloped, axonal projections may be misguided or the β lobes may be fused.

Expression of EcRhs.T:Scer\GAL4 using heat shock results in germaria that are filled with supernumerary germline cells containing a single spectrosome (SSCs) in females. The ratio between developing fusome-containing cells and SSCs in regions 1-2A of the germaria is decreased in the mutants compared to wild type.

Expression of EcRhs.T:Scer\GAL4 using heat shock for 3 to 5 hours after egg laying results in embryonic lethality, with highly penetrant defects in germ band retraction and head involution and defects in cuticle deposition. The embryonic lethality caused by heat treating EcRhs.T:Scer\GAL4/+ embryos at 3 to 5 hours after egg laying is increased from 60% to 96% penetrance if the embryos are also heterozygous for EcRM554fs. The penetrance and expressivity of the germ band retraction defect is also increased in these embryos.

Expression of EcRhs.T:Scer\GAL4 using heat shock in late third instar larvae results in 100% lethality. 62% of animals carrying EcRhs.T:Scer\GAL4 and subjected at 10-12 hours before puparium formation to two sequential heat shocks separated by a 4 hour recovery period arrest development as non-pupariating late third instar larvae, 28% form untanned elongated prepupae and the remaining 10% form partially tanned and misshapen pupal cases. The salivary glands of the non-pupariated larvae fail to undergo programmed cell death in 6 out of 7 cases. The larval midguts are arrested at various stages of cell death, with the gastric caecae often persisting. The imaginal discs fail to undergo metamorphosis. Animals carrying EcRhs.T:Scer\GAL4 and subjected to heat shock during the early pupal stage (12-20 hours after puparium formation) almost all survive.

External Data
Interactions
Show genetic interaction network for Enhancers & Suppressors
Phenotypic Class
Suppressed by
Statement
Reference
Phenotype Manifest In
Enhanced by
Suppressed by
Additional Comments
Genetic Interactions
Statement
Reference

Co-expression of let-7Scer\UAS.cCa rescues the mushroom body α/β lobe defects seen in animals expressing EcRhs.T:Scer\GAL4.

The penetrance and expressivity of the germ band retraction defect seen in embryos in which EcRhs.T:Scer\GAL4 is expressed using heat shock for 3 to 5 hours after egg laying is enhanced by mys1/+ or mys11/+.

Xenogenetic Interactions
Statement
Reference
Complementation and Rescue Data
Comments
Images (0)
Mutant
Wild-type
Stocks (3)
Notes on Origin
Discoverer
Comments
Comments

The EcRhs.T:Scer\GAL4 protein produced can bind to Scer\UAS regulatory sequences in the absence of ligand, but should not activate transcription. In the presence of ligand the protein can switch to an active conformation and induce expression from the Scer\UAS regulatory sequences.

External Crossreferences and Linkouts ( 0 )
Synonyms and Secondary IDs (2)
Reported As
Symbol Synonym
EcRGAL4::LBD.hs
EcRhs.T:Scer\GAL4
Name Synonyms
Secondary FlyBase IDs
    References (13)