Expression of Sox102F^JF02118 results in: microchaetae morphology and distribution defects including misorientation, accumulation of upper dorsal and loss of dorsal centre microchaetae (with the Scer\GAL4^pnr.PU driver), courtship behavior defects, reduced brain size and brain tissue degeneration (with Scer\GAL4^insc.PU) in adults as well as dendritic arbor morphology defects (reduced dendrite length, arbor surface area and its branching complexity) in dendritic arborizing neuron of third instar larvae (with Scer\GAL4^ppk.PU), impaired larval crawling as well as adult climbing abilities along with reduced number of boutons at the larval neuromuscular junctions (with Scer\GAL4^Toll-6-D42) and compromised larval olfactory perception as assessed by fructose preference assay (with the Scer\GAL4[Tab2-201Y] driver).

Flies expressing Sox102F^JF02118 in the heart under the control of Scer\GAL4^how-24B appear normal at eclosion. 30 day old flies exhibit cardiac hypertrophy, a decrease in size of the heart chamber and a decrease in heart rate and wall velocities compared to controls. This is accompanied by disrupted myofibril structure.

Expression of Sox102F^JF02118 in the wing under the control of Scer\GAL4^sd-SG29.1 results in a significant increase in the L2, L3 and wing marginal veins; in particular extra veins are formed in the distal part of L3.