M(3)67C, M(3)i, M(3)i55, minute, M(3)RpS17
Gene model reviewed during 5.45
Low-frequency RNA-Seq exon junction(s) not annotated.
There is only one protein coding transcript and one polypeptide associated with this gene
Click to get a list of regulatory features (enhancers, TFBS, etc.) and gene disruptions (point mutations, indels, etc.) within or overlapping Dmel\RpS17 using the Feature Mapper tool.
GBrowse - Visual display of RNA-Seq signalsView Dmel\RpS17 in GBrowse 2
Please Note FlyBase no longer curates genomic clone accessions so this list may not be complete
Please Note This section lists cDNAs and ESTs that fall within the genomic extent of the gene model, which may include cDNAs and ESTs of genes within introns, or of overlapping genes. Please see GBrowse for alignment of the cDNAs and ESTs to the gene model.
For each fully sequenced cDNA the DGRC maintains various forms of the cDNA (e.g tagged or untagged) in several different host vectors for subsequent cloning and expression in Drosophila and Drosophila cell lines.
Source for merge of: RpS17 BcDNA:RE44119
The genetically defined "M(3)67C" locus (characterized by previous aneuploidy analyses) likely comprises two separable, closely linked Minute genes ("RpS9" and "RpS17").
Source for merge of RpS17 BcDNA:RE44119 was a shared cDNA ( date:030728 ).
Likely Minute gene.
In a sample of 79 genes with multiple introns, 33 showed significant heterogeneity in G+C content among introns of the same gene and significant positive correspondence between the intron and the third codon position G+C content within genes. These results are consistent with selection adding against preferred codons at the start of genes.
A yeast phenotypic screen efficiently identifies conserved genes from more complex organisms and sheds light on their potential in vivo functions. Induced expression of RpS17 proteins causes aberrant cell shapes reflecting defects in cytokinesis and/or cel shape maintenance.
Isolated using a human ribosomal protein S17 cDNA as a probe.
Mutant alleles have the developmental delay and bristle phenotype that makes them useful as markers in clonal analysis.
subdivisions should accompany revised localizations.
One of a class of genes (see MIN record) that when present in one, rather than two, copies, produce a characteristic phenotype consisting of short slender bristles and delayed development. Moderate alleles have good heterozygous viability; extreme alleles very late eclosing <up>45 hr according to Ferrus (1975)</up> with poor viability, and females usually sterile. Viability further reduced in presence of su(f) alleles (Girton, Langner, Cejka, 1986).