Dsec\I-element and Dmau\I-element are more closely related to the chromosomal I-elements of D.melanogaster than to those in any species.
Transformation of Dtei\I-element into D.melanogaster demonstrates that transposition of Dtei\I-element is limited to the female germline, as are I-elements. Hybrid dysgenesis of Dtei\I-element is produced when males carrying more than one copy of Dtei\I-element are crossed with reactive females. The copy number of Dtei\I-element is regulated by the host genome and not by sequences present within Dtei\I-element.
Comparison of active and defective heterochromatic D.melanogaster I-elements with an active Dtei\I-element suggests that the defective heterochromatic I-elements may have become immobilised before the divergence of D.melanogaster and D.teissieri.
A Dtei\I-element has been cloned and sequenced. The element is 5386bp long, and is flanked by an 11bp target-site duplication.
Dsec\I-element and Dmau\I-element are more closely related to the chromosomal I-elements of D.melanogaster than to those in any species.
Transformation of Dtei\I-element into D.melanogaster demonstrates that transposition of Dtei\I-element is limited to the female germline, as are I-elements. Hybrid dysgenesis of Dtei\I-element is produced when males carrying more than one copy of Dtei\I-element are crossed with reactive females. The copy number of Dtei\I-element is regulated by the host genome and not by sequences present within Dtei\I-element.
Comparison of active and defective heterochromatic D.melanogaster I-elements with an active Dtei\I-element suggests that the defective heterochromatic I-elements may have become immobilised before the divergence of D.melanogaster and D.teissieri.
A Dtei\I-element has been cloned and sequenced. The element is 5386bp long, and is flanked by an 11bp target-site duplication.