FB2026_01 , released March 12, 2026
FB2026_01 , released March 12, 2026
Aberration: Dmel\In(3R)E(spl)rv1
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General Information
Symbol
In(3R)E(spl)rv1
Species
D. melanogaster
Name
Inversion (3R) Enhancer of split-revertant
FlyBase ID
FBab0005573
Feature type
Also Known As
E(spl)R1, Df(3R)E(spl)R1, In(3R)E(spl)R1
Computed Breakpoints include

96F2;96F12-96F14;99C

Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Class of aberration (relative to wild type)
Breakpoints

96F2;96F12-96F14;99C

Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

bk1 hits E(spl)

Genetic mapping information
Comments
Comments on Cytology

All limits from polytene analysis (FBrf0047951)

Sequence Crossreferences
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Complementation Data
Completely deleted / disrupted
Partially deleted / disrupted
Molecular Data
Completely deleted
Partially deleted
Genes NOT Deleted / Disrupted
Complementation Data
 
Molecular Data
 
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
 
Molecular Data
 
Affected Genes Inferred by Location (0)
    If no genes are listed here, it may be because the affected region is very large. The JBrowse insert above may show an error for the same reason, and other FlyBase tools such as CytoSearch may also fail for large regions. You can contact FlyBase for more help.
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    Phenotypic Data
    In combination with other aberrations
    NOT in combination with other aberrations

    Strong neurogenic phenotype.

    Not capable of rescuing the lethality conferred NAx-E2/NAx-9 heterozygotes.

    Strong phenotypic reversion: 250--350 facets per eye. Severe embryonic phenotype: loss of ventral, lateral and majority of dorsal cuticle. Viable when heterozygous with a Dl allele.

    Embryonic lethal, extreme neurogenic phenotype.

    Lethal in combination with grounspecified and E(spl)1. Homozygotes have extreme neural hyperplasia and suppress the Nspl-1 phenotype.

    Stocks (1)
    Notes on Origin
    Discoverer
     

    Revertant

    Balancer / Genotype Variants of the Aberration
     
    Separable Components
     
    Other Comments
     

    Identification: Isolated by X-ray mutagenesis of E(spl)1 followed by screening for loss of ability to enhance Nspl-1.

    Synonyms and Secondary IDs (11)
    Reported As
    Symbol Synonym
    Name Synonyms
    Inversion (3R) Enhancer of split-revertant
    Secondary FlyBase IDs
    • FBab0002572
    • FBab0022397
    References (14)